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Journal of Experimental & Clinical Cancer Research : CR logoLink to Journal of Experimental & Clinical Cancer Research : CR
. 2023 Nov 21;42:305. doi: 10.1186/s13046-023-02895-8

Correction: Helicobacter pylori CagA promotes epithelial mesenchymal transition in gastric carcinogenesis via triggering oncogenic YAP pathway

Nianshuang Li 1,2, Yan Feng 2, Yi Hu 1, Cong He 1, Chuan Xie 1, Yaobin Ouyang 1, Stephen C Artim 2, Deqiang Huang 1, Yin Zhu 1, Zhijun Luo 3, Zhongming Ge 2,✉,#, Nonghua Lu 1,✉,#
PMCID: PMC10662552  PMID: 37986030

Correction: J Exp Clin Cancer Res 37, 280 (2018)

https://doi.org/10.1186/s13046-018-0962-5

Following the publication of the original article [1], duplicate images were found in Figure 7b and Figure 7d. The correct figure is given below:

Fig. 7.

Fig. 7

a, b Wound healing assay (a) and Boyden chamber assay (b) were performed in AGS cells infected with H. pylori wild-type strains (PMSS1 or 7.13) and CagA − mutants. c AGS cells were co-cultured with CagA + H. pylori strains PMSS1 or 7.13 in combination with VP treatment, subsequently cells invasion was analyzed by transwell assay. d, e Cell migration were analyzed by wound healing assay (d) and Boyden chamber assay. Data for gene expression are mean ± SEM of 3 independent experiments

Footnotes

Zhongming Ge and Nonghua Lu contributed equally to this work.

Contributor Information

Zhongming Ge, Email: zge@mit.edu.

Nonghua Lu, Email: lunonghua@ncu.edu.cn.

Reference

  • 1.Li N, Feng Y, Hu Y, et al. Helicobacter pylori CagA promotes epithelial mesenchymal transition in gastric carcinogenesis via triggering oncogenic YAP pathway. J Exp Clin Cancer Res. 2018;37:280. doi: 10.1186/s13046-018-0962-5. [DOI] [PMC free article] [PubMed] [Google Scholar]

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