Skip to main content
NIHPA Author Manuscripts logoLink to NIHPA Author Manuscripts
. Author manuscript; available in PMC: 2016 Aug 1.
Published in final edited form as: Dev Psychol. 2015 Jun 8;51(8):1026–1047. doi: 10.1037/dev0000024

The Developmental Costs and Benefits of Children’s Involvement in Interparental Conflict

Patrick T Davies 1, Jesse L Coe 2, Meredith J Martin 3, Melissa L Sturge-Apple 4, E Mark Cummings 5
PMCID: PMC4516573  NIHMSID: NIHMS690884  PMID: 26053147

Abstract

Building on empirical documentation of children’s involvement in interparental conflicts as a weak predictor of psychopathology, we tested the hypothesis that involvement in conflict more consistently serves as a moderator of associations between children’s emotional reactivity to interparental conflict and their psychological problems. In Study 1, 263 early adolescents (M age = 12.62 years), mothers, and fathers completed surveys of family and child functioning at two measurement occasions spaced two years apart. In Study 2, 243 preschool children (M age = 4.60 years) participated in a multi-method (i.e., observations, structured interview, surveys) measurement battery to assess family functioning, children’s reactivity to interparental conflict, and their psychological adjustment. Across both studies, latent difference score (LDS) analyses revealed that involvement moderated associations between emotional reactivity and children’s increases in psychological (i.e., internalizing and externalizing) problems. Children’s emotional reactivity to interparental conflict was a significantly stronger predictor of their psychological maladjustment when they were highly involved in the conflicts. In addition, the developmental benefits and costs of involvement varied as a function of emotional reactivity. Involvement in interparental conflict predicted increases in psychological problems for children experiencing high emotional reactivity and decreases in psychological problems when they exhibited low emotional reactivity. We interpret the results in the context of the new formulation of emotional security theory (e.g. Davies & Martin, 2013) and family systems models of children’s parentification (e.g., Byng-Hall, 2002).

Keywords: interparental conflict, child coping, child emotional reactivity, child psychopathology, emotional security


Children’s involvement in conflicts between their parents has long been regarded as a risk factor for psychological maladjustment (Davies & Cummings, 1994; Emery, 1989; Johnston, Roseby, & Kuehnle, 2009). Consistent with this thesis, a meta-analysis of over a dozen studies indicated that children’s involvement in interparental conflict was a significant correlate of their internalizing and externalizing symptoms (Rhoades, 2008). By the same token, the modest to moderate strength of these associations highlights the need to better understand the role children’s involvement plays in their coping and adjustment. According to family process models (e.g., Byng-Hall, 2002; Cummings & Davies, 1996), children’s involvement is an inextricable part of understanding the nature and developmental sequelae of their broader constellations of responding to parental difficulties. Therefore, our primary objective in this paper is to examine whether involvement may function more consistently as a moderator of associations between children’s distress responses to interparental conflict and their psychological difficulties. More specifically, we test the hypothesis that the multiplicative interaction between involvement and distress is a consistent predictor of children’s internalizing and externalizing symptoms even after the inclusion of each of these forms of responding to conflict as additive predictors.

Although several conceptualizations share the assumption that involvement in interparental conflict is an important component in understanding how children cope in high conflict homes (e.g., Crockenberg & Langrock, 2001; Davies & Cummings, 1994; Grych & Fincham, 1990), involvement is most prominently featured in emotional security theory (EST; Davies & Cummings, 1994). According to EST, children’s involvement in interparental conflict is a central barometer for difficulties in preserving a sense of emotional security in the interparental relationship. Interpreted within this model, children’s efforts to become involved in parental conflicts reflect their attempts to exert control over parental emotionality and behavior before disputes can escalate and result in more serious, negative consequences. However, children’s efforts to serve as protectors, confidantes, or emotional caretakers of parents in the midst or aftermath of conflict require considerable forethought, emotional investment, and personal risk. Thus, EST proposes that protracted involvement in parental disputes over time increases children’s risk for developing internalizing and externalizing symptoms through several mechanisms. For example, high levels of involvement are proposed to reflect the disproportionate allocation of psychobiological resources toward personal safety at the expense of investing in other significant goals necessary for successful adaptation (e.g., mastery of physical and social environment). The progressive accumulation of difficulties in resolving multiple developmental challenges, in turn, may broadly increase children’s risk for experiencing psychopathology. Likewise, high levels of involvement are commonly accompanied by preoccupation, helplessness, distress, and loyalty pulls with parents and, as a result, may set the stage for more pervasive adjustment problems (Davies, 2002).

Family systems theory converges with EST in proposing that forms of involvement in the interparental relationship that reflect emotional preservation or enmeshment incur significant psychological costs (e.g., Byng-Hall, 2002; Jurkovic, Morrel, & Thirkfield, 1999; Minuchin, 1974). These models maintain that distinguishing between harmful and benign forms of involvement in adverse family circumstances requires analysis of the substance and quality of children’s involvement strategies. Benign forms of involvement are defined by mild forms of child caretaking behavior that maintain the child’s distinct identity and autonomy. Conversely, developmentally destructive types of “parentification” involve excessive emotional and psychological entanglement. The resulting dependency and enmeshment in interparental difficulties are further proposed to increase children’s vulnerability for subsequent psychological problems.

Taken together, family systems theory and EST share the assumption that more precisely understanding the developmental implications of children’s involvement in conflict requires distinguishing between different forms of involvement based on whether or not they carry significant personal risk and emotional investment. Guided by these models, research has differentiated between two dimensions of involvement: mediation and coercion. Mediation, which is also known in the literature as “involvement” (Buehler, Lange, & Franck, 2007), agentic involvement (Schermerhorn, Chow, & Cummings, 2010; Schermerhorn, Cummings, DeCarlo, & Davies, 2007), facilitative behavior (Davis, Hops, Alpert, & Sheeber, 1998), and proactive mediation (Shelton & Harold, 2008), is characterized by relatively benign involvement behaviors manifested in helping, intervening, refereeing, and comforting parents. Conversely, coercive involvement, which is synonymously referred to as aggressive involvement (Davis et al., 1998), dysregulated involvement (Schermerhorn et al., 2010), and overinvolvement (Shelton & Harold, 2008) in the literature, is specifically comprised of intervention behaviors carrying high psychological burden and risk in the form of protecting a parent, siding with one parent against the other, and confronting or arguing with parents.

Although research identifying more precise forms of involvement is still in its early stages, empirical evidence is mixed on whether the concept of coercive involvement yields incremental leverage in identifying children most at risk for developing psychological problems. For example, Shelton and Harold (2008) reported that coercive involvement (i.e., overinvolvement), but not mediation, was a predictor of increases in children’s internalizing and externalizing problems. However, other studies have failed to document any relationship between coercive forms of involvement and children’s psychological problems in cross-sectional and prospective designs (e.g., Buehler et al., 2007; Davies & Cummings, 1998). Still other research has shown that the role of coercive involvement as a predictor of changes in children’s adjustment was modest in magnitude and limited to a specific informant (i.e., child) or outcome (i.e., externalizing problems) (Davis et al., 1998; Jouriles, Rosenfield, McDonald, & Mueller, 2014). Thus, although disaggregating involvement into multiple dimensions may provide some traction in better characterizing children’s risk for psychological problems, the inconsistent findings from this research highlight the importance of adopting complementary approaches.

Our primary thesis is that the discrepancies in findings across studies reflect that children’s involvement in interparental difficulties is an inextricable part of a broader pattern of responding to conflict. Previous approaches to documenting associations between children’s emotional insecurity and psychological adjustment have largely examined involvement as an additive predictor or as part of a linear composite with other forms of reactivity to conflict. At a theoretical level, this approach is at odds with the organizational perspective of EST and its assumption that the developmental meaning and implications of any single form of reactivity varies as a function of children’s other dimensions of responding to interparental conflict (Cummings & Davies, 1996). Thus, against the backdrop of linear, additive approaches to assessing involvement, the present paper is designed to break new ground by examining the synergistic interplay between the two prevailing forms of involvement (i.e., coercion, mediation) and emotional reactivity in predicting children’s psychological problems.

EST provides firm bases for examining emotional reactivity to interparental conflict as a critical context for understanding the multivariate role of involvement. As with involvement, children’s concerns about emotional security are proposed to be expressed through high levels of emotional reactivity that are comprised of intense, prolonged fearful and distressed reactions to interparental conflict. With its strong conceptual emphasis on children’s emotionality in the context of interparental conflict, EST frames children’s emotional reactivity as the cornerstone of the goal-corrected system of emotional security (Davies, Winter, & Cicchetti, 2006). In accord with this conceptualization, the EST component of emotional reactivity is a consistent predictor of children’s psychological problems across a wide array of methodological approaches (e.g., naturalistic, experimental), designs (e.g., cross-sectional, longitudinal), and analytic models (e.g., multivariate) (e.g., Buehler et al., 2007; Davies, Cicchetti, & Martin, 2012; Davies & Cummings, 1998). Based on the central role emotional reactivity plays in defining security, we specifically investigate whether coercive involvement and mediation may function as vulnerability or susceptibility factors that moderate or alter the well-established association between children’s emotional reactivity and children’s internalizing and externalizing symptoms.

Although questions remain about precisely how children’s forms of involvement may moderate the risk posed by emotional reactivity, the translation of developmental psychopathology models to EST yields three prevailing classes of models: vulnerable-stable, vulnerable-reactive, and vulnerable-adaptive. Each of these models is explicitly designed to offer a comprehensive characterization of involvement as a moderator by examining both dimensions of the interaction (see Luthar, Cicchetti, & Becker, 2000; Roisman et al., 2012). In fully delineating involvement as a moderator of emotional reactivity, the models vary along the dimension of how involvement alters associations between emotional reactivity and psychopathology. By the same token, guidelines for distinguishing between the three models in the literature also call for the analysis of whether associations between children’s involvement and their psychological problems vary at high and low levels of emotional reactivity.

In drawing on the developmental psychopathology taxonomy of vulnerability effects (Luthar et al., 2000), the vulnerable-stable model shown in Figure 1a proposes that responding to conflict characterized by high involvement, emotional reactivity, or both are comparable in the degree to which they increase children’s vulnerability to psychological problems. Thus, the combination of emotional reactivity and involvement does not incrementally increase children’s disadvantage above and beyond either form of reactivity considered singly. As denoted by the solid line in the figure, high levels of involvement specifically take a significant and stable psychological toll on children regardless of the level of emotional reactivity they experience during interparental conflicts. Thus, children’s emotional reactivity in this model only predicts increases in their internalizing and externalizing symptoms when their involvement in conflict is low (see the dotted line in Figure 1a). Likewise, due to the mutual potency of involvement and emotional reactivity risk factors, involvement is proposed to predict higher levels of children’s psychological problems only when emotional reactivity is low. Although no studies have examined the interplay between emotional reactivity and forms of involvement, the moderating role of coercive involvement may be more likely than mediation to assume a vulnerable-stable form. More specifically, if coercive involvement is a more potent risk factor than mediation, as some empirical findings indicate (e.g., Davis et al., 1998; Shelton & Harold, 2008), it is plausible that both emotional reactivity and coercion would predict children’s problems when the other form of responding to conflict is low.

Figure 1.

Figure 1

Figure 1

Figure 1

a. Conceptual illustration of the vulnerable-stable form of moderation.

b. Conceptual illustration of the vulnerable-reactive form of moderation.

c. Conceptual illustration of a vulnerable-adaptive form of moderation.

In the second conceptualization of moderation shown in Figure 1b, the vulnerable-reactive model proposes that high involvement amplifies predictive pathways between children’s emotional reactivity and their psychological difficulties (Luthar et al., 2000). Whereas emotional reactivity is a relatively weak predictor of changes in children’s psychological problems at low levels of involvement (see dotted line in Figure 1b), it may take an exponentially greater toll on children when they are heavily involved in interparental problems (see solid line in Figure 1b). A primary premise is that the greater psychological immersion underlying children’s involvement in interparental conflict magnifies the impact of children’s emotional reactivity to parental disagreements on their psychological adjustment problems. In a corresponding fashion, the vulnerable-reactive model also hypothesizes that children’s extensive involvement in interparental conflict only carries significant psychological costs when it is organized by high levels of emotional reactivity. Without an existing empirical base to guide predictions, it is premature to formulate any firm hypotheses on the viability of identifying the vulnerable-reactive form of moderation. However, because mediation and coercion are each characterized by some level of emotional entanglement in conflicts (Buehler et al., 2007; Davies & Cummings, 1998; Harold & Shelton, 2008), it is possible that associations between children’s emotional reactivity and their psychopathology could be magnified in a vulnerable-reactive way in the presence of either form of involvement.

As the third conceptualization of involvement as a moderator, the vulnerable-adaptive model proposes that the meaning of children’s involvement shifts drastically from carrying psychological disadvantages at high levels of emotional reactivity to conferring significant adaptive benefits at low levels of emotional reactivity. Thus, as shown by the steep slope of the solid line in Figure 1c, the vulnerable-adaptive and vulnerable-reactive models share the hypothesis that associations between emotional reactivity and psychopathology will be particularly evident when children are highly involved in their parents’ conflicts. However, as the central source of distinction between the two models, the vulnerable-adaptive model proposes that children exhibiting higher levels of involvement in parental conflicts may actually fare substantially better in their psychological functioning when they experience low levels of emotional reactivity. Although the form of the moderation is comparable to the interaction proposed by differential susceptibility theory (Belsky & Pleuss, 2009), the mechanisms postulated to underlie the effect are different. Thus, when children experience heightened emotional reactivity to interparental conflict, high levels of involvement reflect their prolonged difficulties preserving emotional security and, as a consequence, disproportionately increase their vulnerability to psychological problems (Davies, 2002). Conversely, under conditions of low emotional reactivity (e.g., low fear, distress, and vigilance), involvement is theorized to be motivated by well-regulated, empathetic concern for the welfare of their parents. Accordingly, involvement coupled with low emotional reactivity may have benign ramifications for children’s mental health (Davies & Martin, 2013). Vulnerable-adaptive models presuppose that there are null associations between the proposed moderator (i.e., mediation, coercive involvement) and outcomes (Belsky, Bakermans-Kranenburg, & van IJzendoorn, 2007). Therefore, by virtue of its proposed innocuous implications for children’s development, mediation may be particularly likely to assume the moderating role proposed in the vulnerable-adaptive model. However, due to inconsistencies in the identification of coercion and mediation as risk factors (Buehler et al., 2007; Davies & Cummings, 1998), it is also plausible that the both forms of involvement may evidence vulnerable-adaptive moderating effects.

In summary, the present article is the first, to our knowledge, to test the theoretically guided hypothesis that the multiplicative interplay between children’s emotional reactivity and involvement in the face of interparental conflict uniquely predicts their psychological problems. Children’s internalizing and externalizing symptoms not only have substantial societal implications but are the most commonly investigated correlates and sequelae of their reactivity to interparental conflict (e.g., Rhoades, 2008). Therefore, to afford a base of comparison with the existing knowledge, we examine these two forms of psychological problems as outcomes of the interaction between emotional reactivity and involvement. To provide an authoritative test of the replicability of our findings, we capitalize on data drawn from two large studies (n > 240) of children and their families that vary in their measurement techniques, methodological designs, and developmental characteristics of the children.

Study 1

As a first step to addressing our aims, Study 1 examined the synergistic interplay between children’s emotional reactivity and involvement as predictors of subsequent change in psychological maladjustment for young adolescents. Early adolescence is a highly significant developmental period for understanding children’s coping with interparental conflict. Research supports the premise that adolescence is a period of heightened sensitivity to interparental problems (e.g., Buchanan, Maccoby, & Dornbusch, 1991; Cummings, Schermerhorn, Davies, Goeke-Morey, & Cummings, 2006). Although no studies, to our knowledge, have examined age differences in children’s forms of behavioral involvement over developmental periods that include adolescence, teens have been shown to experience stronger impulses to mediate conflicts in comparison to younger children (Cummings, Ballard, El-Sheikh, & Lake, 1991; Davies, Myers, & Cummings, 1996). Given that both parents and children report that the effectiveness of interventions diminishes substantially from childhood into adolescence (Covell & Miles, 1992), heightened involvement in conflicts may do little more than embroil adolescents in protracted discord between parents and, in the process, increase their risk for psychological problems.

Relative to earlier developmental periods, dimensions of sensitivity to conflict are also theorized to increase in adolescence by virtue of teens’ greater awareness of subtle interparental difficulties and longer histories of exposure to interparental conflict (Cummings et al., 2006; Davies et al., 1999). Increases in the temporal stability in patterns of reactivity to family adversity in conjunction with concomitant rises in developmental (e.g., autonomy from parents, identity development) and contextual (e.g., peak occurrence of stressful life events) challenges during adolescence may further amplify their vulnerability to psychological problems (e.g., Davies & Cummings, 2006; Rhoades, 2008). Consistent with this conceptualization, metaanalytic findings revealed that associations between children’s involvement and emotional reactivity and their internalizing and externalizing symptoms were significantly stronger during adolescence in comparison to childhood (Rhoades, 2008).

To rigorously address our objective, we utilized a multi-informant (i.e., adolescents, mothers, and fathers) measurement battery nested within a longitudinal design. We selected a two-year time window between measurement occasions based on empirical and conceptual considerations. At an empirical level, multiple laboratories examining the sequelae of adolescent responses to interparental conflict have examined changes in psychological problems over two year periods (e.g., Buehler et al., 2007; Cummings et al., 2006; Shelton & Harold, 2008). At a conceptual level, EST proposes that children’s responses to interparental conflict set in motion the emergence of risk mechanisms that, over months or years, gradually intensify, broaden, and crystallize into the development of children’s psychological problems (e.g., Davies et al., 2006). This unfolding sequence of processes is theorized to take place over a longer period (i.e. years rather than months) during adolescence than earlier periods by virtue of progressive constraints in the plasticity of functioning during the teen years (Frankenhuis & Del Giudice, 2012).

Methods

Participants

Data for this study were drawn from a longitudinal project on family relationship processes and adolescent development. Participants in the larger study consisted of 280 families with adolescents who were recruited through local school districts and community centers in a moderate-sized metropolitan area in the Northeast and a small city in the Midwest. However, due to our aim of examining children’s responses to interparental conflict, families were only included in this paper if the mothers, fathers, and adolescents had regular contact with each other. Regular contact was specifically defined as maintaining contact as a triad for an average of 2 to 3 days per week during the year. Instituting this criterion resulted in the exclusion of 17 families from this paper, yielding a sample of 263 mothers, fathers, and adolescents. Participants returned two years later to participate in the second wave of data collection. The retention rate across the two-year period was 85%. The average age of adolescents at Wave 1 was 12.62 years (SD = .57), with 50% of the sample consisting of girls. Median household income of the families was between $55,000 and $74,999 per year. Median education level of mothers and fathers was between some college education and an Associate’s degree. Most parents (i.e., 89%) were married at the outset of the study. The majority of the adolescents were White (74%), followed by smaller percentages of African American (17%), multi-racial (8%), and other races (1%). Approximately 4% of the adolescents were Latino. Adolescents lived with their biological mother in most cases (93%), with the remainder living with an adoptive or stepmother (4%) or a female guardian (3%). In addition, children lived with their biological father in the majority of cases (79%), with the remainder of the sample living with either an adoptive or stepfather (16%) or a male guardian (5%).

Procedures and Measures

Families visited the laboratory at one of the two research sites. During the first wave, adolescents completed survey measures to assess their responses to interparental conflict, and their mothers filled out demographic and family background information. At each wave, mothers, fathers, and children completed questionnaires to assess children’s internalizing and externalizing symptoms. All research procedures were approved by the Institutional Review Board at both research sites prior to conducting the study. Families were compensated monetarily for their participation.

Adolescent responses to interparental conflict

Adolescents completed three scales derived from the Security in the Interparental Subsystem (SIS) scales to assess their emotional reactivity and involvement (Davies, Forman, Rasi, & Stevens, 2002). Children’s emotional reactivity was specifically indexed by children’s reports on the Emotional Reactivity scale. The scale consists of nine items designed to capture children’s multiple, prolonged fear and distress in response to conflict (e.g., “When my parents argue, I feel scared”). The newer, longer version of the SIS contains two involvement scales that distinguish between mediation and coercion (see Shelton & Harold, 2008). The “Mediation” scale consists of six items assessing children’s efforts to intervene in the conflict through comforting (e.g., “I try to comfort one or both of them”), distracting (e.g., “I try to distract them by bringing up other things”), solving the problem (e.g., “I try to solve the problem for them”), or directly interceding (e.g., “I tell them to stop fighting”). In contrast, the SIS Coerciveness scale, which is also referred to as the Overinvolvement scale (Shelton and Harold, 2008), contains five items that capture bossy, aversive, and domineering approaches to interrupting parental conflicts (e.g., “I argue with one or both of them,” “I end up taking sides with one of them,” “I tell one of my parents that he or she is wrong”). Response alternatives for the SIS items were as follows: 1 = Not at all true of me; 2 = A little true of me; 3 = Somewhat true of me; and 4 = Very true of me. To confirm the factor structure of the items in the two involvement scales, we conducted a principal components analysis with varimax rotation. The results supported a two-factor solution (e.g., eigenvalues were greater than 1.0 for the two factors) that discriminated between the items on the Coerciveness and Mediation subscales. Alpha coefficients were .89, .75, and. 74 for the Emotional Reactivity, Coerciveness, and Mediation scales, respectively. The validity of the scales is supported by their concurrent and prospective associations with children’s exposure to family conflict and their psychological adjustment (e.g., Davies, Sturge-Apple, Bascoe, & Cummings, 2014; Shelton & Harold, 2008).

Adolescent internalizing and externalizing symptoms

To obtain multi-informant assessments of internalizing and externalizing symptoms, mothers, fathers, and adolescents completed child psychopathology questionnaires. Mother and father reports of adolescent psychological problems were derived from the Internalizing (e.g., “Nervous, high-strung, or tense,” “Unhappy, sad, or depressed”) and Externalizing (e.g., “Temper tantrums or hot temper,” “Gets in many fights”) scales from the Child Behavior Checklist (CBCL; Achenbach, Dumenci, & Rescorla, 2003). Previous studies have provided consistent evidence for the reliability and convergent and discriminant validity of the CBCL scales (e.g., Achenbach et al., 2003). Consistent with recommendations, we used the sum of raw scores on the CBCL scales in the analyses. Alpha coefficients indexing internal consistency ranged from .82 to .91 for parents across the two waves. In addition, children completed the Conduct Problems (5 items; e.g., “I fight a lot”) and Emotional Symptoms (5 items; e.g., “I worry a lot”; “I am often unhappy, downhearted, and tearful”) scales from the Strengths and Difficulties Questionnaire (SDQ; Goodman, 1999) to obtain assessments of externalizing and internalizing symptoms, respectively. The psychometric properties of the two SDQ scales have been supported in previous studies (e.g., Goodman, 1999). Internal consistencies for the two scales across the two waves ranged from .64 to .74. The three-informant approach specifically permitted use of mother, father, and child reports as manifest indicators of latent constructs of internalizing and externalizing symptoms at each wave.

Covariates

Two covariates were derived from parent reports of demographic background characteristics: (1) children’s gender (1 = boys; 2 = girls) and (2) total annual family income based on a 13-point ordinal scale ranging from 1 (less than $6,000) to 13 ($125,000 or more). As the third and final covariate, we assessed destructive interparental conflict based on mother and father reports on three scales. Mother and fathers completed the 10-item O’Leary Porter Scale to assess children’s exposure to marital hostility (OPS; Porter, & O’Leary, 1980; e.g., “How often do you and/or your partner display verbal hostility [raised voices, etc.] in front of your child?”). The remaining scales, which were derived from the Conflict and Problem-Solving Scales (CPS; Kerig, 1996), reflected the frequency with which parents and their partners engaged in (a) physically violent acts (e.g., Physical Aggression; e.g., “Push, pull, shove, grab partner”) and (b) impasses in conflict characterized by unresolved hostility, distress, and disengagement (e.g., Stonewalling; e.g., “Storm out of the house”). Internal consistencies for the maternal and paternal reports on the four scales ranged from .78 to .91. The six scales were standardized and aggregated to form a single, parsimonious composite of interparental conflict (α = .81).

Results

Descriptive and Preliminary Analyses

For descriptive purposes, Table 1 provides the means, standard deviations, and correlations for primary variables in the study. Inspection of the means for individual scales of the interparental conflict composite revealed means that were similar to community samples of families in previous studies. For example, means on the CPS Physical Aggression (M = 2.11, SD = 3.60) and Verbal Aggression (M = 22.92, SD = 9.21) scales were comparable to levels identified in prior working and middle class samples of families (CPS Physical Aggression Means ranging from 2.06 to 2.14; CPS Verbal Aggression Means ranging from 22.15 to 23.87; Crockenberg, Leerkes, & Lekka, 2006 ; El-Sheikh, Harger, & Whitson, 2001; Fosco & Grych, 2007; McConnell & Kerig, 2002).

Table 1.

Means, Standard deviations, and Correlations for the Primary Variables in the Study 1 Analyses.

Mean SD 1 2 3 4 5 6 7 8 9 10 11 12 13 14 15 16 17
Wave 1 Covariates
 1. Child Gender 0.50 0.50
 2. Household Income 8.47 3.14 −.06
 3. Interparental Conflict 0.00 0.71 .03 −.22*
Wave 1 Child Reactivity to Conflict
 4. Emotional Reactivity 14.63 5.61 .09 −.15* .20*
 5. Mediation 12.48 3.97 .06 −.10 .14* .34*
 6. Coerciveness 6.36 2.23 −.04 −.17* .17* .35* .30*
Wave 1 Internalizing Symptoms
 7. Paternal CBCL 5.58 5.10 .05 −.13* .22* .15* .02 .12
 8. Maternal CBCL 5.87 5.43 .11 −.10 .24* .02 −.05 −.01 .42*
 9. Child SDQ 2.12 2.23 .24* −.19* .13* .41* .05 .26* .26* .25*
Wave 1 Externalizing Symptoms
 10. Paternal CBCL 5.65 6.53 −.08 −.26* .13* .07 .02 .30* .52* .26* .22*
 11. Maternal CBCL 5.62 6.14 −.13* −.16* .19* .03 −.11 .23* .31* .48* .23* .66*
 12. Child SDQ 1.79 1.82 −.10 −.22* .08 .22* .00 .35* .17* .09 .46* .53* .45*
Wave 2 Internalizing Symptoms
 13. Paternal CBCL 4.51 5.38 .09 −.22* .21* .09 −.07 .11 .63* .25* .26* .42* .32* .17*
 14. Maternal CBCL 4.78 4.82 .16* −.18* .18* .04 −.04 .12 .37* .61* .19* .29* .44* .09 .42*
 15. Child SDQ 2.33 2.32 .34* −.16* .11 .15* .06 .00 .24* .25* .46* .11 .04 .11 .26* .29*
Wave 2 Externalizing Symptoms
 16. Paternal CBCL 4.93 6.28 −.07 −.23* .15* .17* −.01 .39* .51* .13 .22* .74* .60* .39* .62* .30* .07
 17. Maternal CBCL 4.76 5.55 −.08 −.25* .12 .14* −.04 .39* .31* .27* .26* .59* .76* .45* .32* .47* .08 .70*
 18. Child SDQ 1.84 1.80 −.12 −.32* .13 .21* −.01 .26* .14* .07 .18* .35* .34* .35* .20* .17* .20* .37* .48*

Note. Child gender: 1= boys; 2 = girls. p ≤ .05.

To index change in adolescent internalizing and externalizing problems, we used latent difference score (LDS) modeling across the two measurement occasions. LDS offers a powerful way of testing whether the interaction predicts interindividual differences in intraindividual changes in adjustment over time through the specification of latent change scores (McArdle, 2009). Because our goal of identifying predictors of adolescent psychological problems first requires demonstrating that there are significant individual differences in intraindividual change in their symptoms, we tested this assumption by specifying successive unconditional LDS models for internalizing and externalizing symptoms following procedures for testing strong factorial variance (Widaman, Ferrer, & Conger, 2010). Thus, as Figure 2 depicts, measurement equivalence across time was maximized by placing the following constraints in the analyses: (1) fixed and free factor loadings are identical over time; (2) factor loadings of each of the indicators of internalizing and externalizing symptoms were constrained to be equal across time; and (3) intercepts of the same indicators were fixed to be invariant across time. Both models fit the data well: χ2 (9, N = 263) = 15.72, p = .07, RMSEA = .05, CFI = 0.98, and χ2/df ratio = 1.75, for internalizing symptoms; and χ2 (9, N = 263) = 14.59, p = .10, RMSEA = .05, CFI = .99, and χ2/df ratio = 1.62, for externalizing symptoms. Moreover, statistically significant variances of 7.31 (z = 5.07) for externalizing symptoms and 5.99 (z = 3.18) for internalizing symptoms indicated that there were significant individual differences in mean changes in adolescent problems from Time 1 to Time 2. Therefore, given our aim of examining why teens differ in their psychological problems over time, we proceeded to our primary analytic goal of examining the interplay between adolescent emotional reactivity and involvement as predictors of changes in their internalizing and externalizing symptoms.

Figure 2.

Figure 2

A conceptual illustration of the specification of unconditional latent difference score models. τ and λ denote factor loadings and intercepts, respectively. λ11 and λ21 are fixed to 1. Loading constraints include λ21 = λ22 and λ31 = λ32. Intercept invariance is as follows: τ11 = τ12; τ21 = τ22; and τ31 = τ32.

Primary Analyses

To test coerciveness and mediation as moderators of associations between adolescents’ emotional reactivity and change in internalizing and externalizing symptoms, four analytic models were conducted based on consecutively pairing emotional reactivity with the two forms of involvement in predicting LDS change for internalizing and externalizing symptoms separately (i.e., 1 emotional reactivity predictor × 2 forms of involvement × 2 outcomes). For each model, we examined emotional reactivity, the specific form of involvement, and their multiplicative product as predictors of LDS change in adolescent psychopathology over the two-year lag in measurement occasions using structural equation modeling (SEM). Emotional reactivity, mediation, and coercion were centered to reduce multicollinearity in the moderator analyses. In addition, we included exposure to interparental conflict, child gender, and family income as covariates in the prediction of change in adolescent psychological symptoms. The same strong factorial invariance constraints in the unconditional LDS were specified in the primary analyses (Widaman et al., 2010). In addition, correlations were also specified between: (a) the primary predictors (i.e., emotional reactivity and involvement), (b) the covariates, (c) the covariates and primary predictors, (d) the Wave 1 latent construct of adolescent psychological functioning and each of the covariates and predictors, and (e) the same informant’s measure of psychological difficulties across the two time points. Finally, consistent with analytic recommendations in LDS approaches, an autoregressive path was specified between the Wave 1 latent construct of adolescent psychological functioning in each model and the latent change in functioning from Wave 1 to Wave 2 (McArdle, 2009). SEM analyses were conducted using full-information maximum likelihood (FIML) in Amos 22.0 to estimate missing data (i.e., data were missing for 10.0% of the values) and retain the full sample for primary analyses (Enders, 2001).

Internalizing symptoms

The two models examining the moderating roles of adolescents’ forms of involvement in analyses of change in their internalizing symptoms provided satisfactory to marginal representations of the data: χ2 (31, N = 263) = 79.85, p < .01, RMSEA = .08, χ2/df ratio = 2.58, and CFI = .90 for the mediating type of involvement; and χ2 (31, N = 263) = 108.86, p < .01, RMSEA = .10,χ2/df ratio = 3.48, and CFI = .87 for the coercive form of involvement. In support of the measurement models, the loadings of the manifest indicators onto the Wave 1 and 2 latent constructs of internalizing symptoms were all significant (p < .001) and moderate to strong in magnitude (ranging from .47 to .65). Further analysis of the structural model revealed that none of the structural paths running from the primary predictors to latent change in internalizing symptoms were significant. Therefore, for succinctness, the findings from the model are not presented.

Externalizing symptoms

SEM analyses testing adolescent mediation in parental conflicts as a moderator of the association between their emotional reactivity and change in their externalizing problems provided a good fit with the data: χ2 (33, N = 263) = 53.20, p < .05, RMSEA = .048, χ2/df ratio = 1.61, and CFI = .97. As shown in Figure 3, the model for adolescent externalizing symptoms yielded significant findings for the predictor and interaction. Consistent with prior research, adolescent emotional reactivity predicted increases in externalizing symptoms over time, β= .19, p < .05. Pertinent to the study hypotheses, the interaction between adolescent emotional reactivity and mediation was also significantly associated with LDS increases in externalizing symptoms over the two-year period, β = .22, p < .01.

Figure 3.

Figure 3

The Study 1 LDS model of adolescents’ mediation and emotional reactivity to interparental conflict as predictors of change in their externalizing symptoms over a two-year period. Significant structural paths are bolded. For clarity, only significant correlations among predictors and covariates are included in the figure. * p < .05.

The moderating effect was first clarified by graphically plotting and calculating simple slopes of emotional reactivity at high (+1 SD) and low (−1 SD) levels of adolescents’ mediation in parental conflicts. To portray the interaction across a relatively comprehensive range of the proposed predictor (i.e., 95%), we conducted simple slope plots and calculations at −2 SD and + 2 SD from the centered mean of emotional reactivity (see Roisman et al., 2012). Findings from the simple slope analyses revealed that emotional reactivity predicted increases in adolescent externalizing symptoms at high, b = 0.21, p < .001, but not low levels, b = −0.01, p = .85, of mediation. As depicted in Figure 5a, the graphical plot of the moderating effects revealed a disordinal (i.e., cross-over) interaction. Although the simple slope analyses and visual inspection of the graphical plot correspond most closely with the vulnerable-adaptive model (see Figure 1c), they do not provide a definitive analysis of its explanatory power relative to the vulnerable-reactive model. Both of these models posit that emotional reactivity is a stronger predictor of psychopathology when mediation is high. Moreover, in spite of the visual similarities between the graphical plot and the interaction proposed in the vulnerable-adaptive model, Roisman and colleagues (2012) have cautioned against using graphical plots to examine whether moderator results are more consistent with models proposing an ordinal interaction (i.e., vulnerable-reactive) or a disordinal interaction (i.e., vulnerable-adaptive).

Figure 5.

Figure 5

Figure 5

a. A graphical plot of the interaction between adolescent emotional reactivity and mediation in predicting LDS change in their externalizing symptoms over a two-year period.

b. A graphical plot of the interaction between adolescent emotional reactivity and coerciveness in predicting LDS change in their externalizing symptoms over a two-year period.

To more authoritatively test which of the moderating patterns proposed in the vulnerable-adaptive and vulnerable-reactive models more closely corresponds with the data, statistical guidelines call for calculating regions of significance on X (RoS on X) tests (Dearing & Hamilton, 2006; Kochanska, Sanghag, Barry, & Philibert, 2011; Roisman et al., 2012). RoS on X tests specifically invert the predictor and moderator to yield analyses of the significance of the association between the moderator and outcome within the bounded regions of the proposed predictor (i.e., + or − 2 SDs). Accordingly, the RoS on X test was used to test whether mediation significantly predicted adolescent externalizing symptoms at high and low levels of their emotional reactivity (Roisman et al., 2012). Support for the vulnerable-reactive model would be evidenced by findings indicating that mediation was significantly associated with increases in adolescent psychopathology only at high levels of emotional reactivity. In contrast, the vulnerable-adaptive model would be supported if the results revealed that mediation was a predictor of significant increases in externalizing problems at high levels of emotional reactivity and significant decreases in externalizing problems at low levels of emotional reactivity. Consistent with the vulnerable-adaptive form of moderation, the results indicated that adolescent mediation in interparental conflict predicted increases in externalizing symptoms at high levels (+2 SD) of emotional reactivity, b = 0.30, p < .05, and reductions in externalizing symptoms at low (− 2 SD) levels of emotional reactivity, b = −0.33, p = .01.

By relying on null hypothesis testing, the results of the RoS on X test vary as a function of the sample size. Small sample sizes may specifically produce low power to detect moderating effects proposed in the vulnerable-adaptive mode. Conversely, large samples run the risk of producing findings in favor of the vulnerable-adaptive pattern that have little substantive significance. Thus, as a sample-independent index for testing the relative fit of the interaction with the vulnerable-adaptive and vulnerable-reactive models, we followed statistical recommendations and calculated the proportion of interaction (PoI) index (e.g., Davies et al., 2014; Roisman et al., 2012). The PoI index consists of the ratio of improved functioning (i.e., decreases in externalizing symptoms) for adolescents with high levels of mediation relative to the overall aggregate of improved and impaired outcomes. If mediation confers no significant advantages for adolescents, as the vulnerable-reactive model proposes, then the PoI index should approach .00 (i.e., between .00 and .16). Conversely, the ratio (i.e., PoI index) should be higher than .16 if the findings support the vulnerable-adaptive proposal that beneficial outcomes are common for adolescents exhibiting high levels of mediation. Strong support for the vulnerable-adaptive model would be evidenced by PoI values between .40 and .60. In support of the vulnerable-adaptive hypothesis, the PoI index for mediation was .54.

Figure 4 depicts the results of the SEM tests of adolescent coerciveness as a moderator of associations between their emotional reactivity and their changes in externalizing symptoms. The model provided a good fit with the data: χ2 (33, N = 263) = 66.66, p < .01, RMSEA = .062, χ2/df ratio = 2.01, and CFI = .96. In accord with the hypotheses, the interaction between adolescent emotional reactivity and coerciveness significantly predicted LDS increases in their externalizing symptoms over the two-year period, β = .32, p < .001. The graphical plot of the moderating effect in Figure 5b revealed a disordinal interaction similar in shape to the moderating effect of adolescent mediation. Simple slope analyses further indicated that adolescents’ emotional reactivity predicted increases in their externalizing symptoms at high, b = 0.20, p < .001, but not low levels, b = −0.04, p = .50, of coerciveness. To quantitatively compare the utility of the moderating effects proposed in the vulnerable-adaptive and vulnerable-reactive models, we also conducted the RoS on X and PoI tests. Consistent with the vulnerable-adaptive model, adolescent coercive involvement predicted increases in their externalizing symptoms at high levels of emotional reactivity, b = 0.67, p < .001, and decreases in their externalizing symptoms at low levels of emotional reactivity, b = −0.57, p = .05. In addition, the PoI coefficient of .42 revealed strong support for the vulnerable-adaptive model and its hypothesis that there is a relatively equal balance of healthy and unhealthy outcomes for teens exhibiting high coerciveness across the range of the emotional reactivity variable.

Figure 4.

Figure 4

The Study 1 LDS model of adolescents’ coercive involvement and emotional reactivity to interparental conflict as predictors of change in their externalizing symptoms over a two-year period. Significant structural paths are bolded. For clarity, only significant correlations among predictors and covariates are included in the figure. * p < .05.

Study 2

Although the Study 1 findings showed that teen involvement served as a moderator of prospective associations between their emotional reactivity and externalizing symptoms, testing the bounds of generalizability and specificity of the results is advanced through replication in a study that differs systematically in its approach. Accordingly, Study 2 was designed to provide a complementary test of the multiplicative interplay between emotional reactivity and involvement by varying methodological and conceptual characteristics. More specifically, we rigorously tested the generalizability of the findings from the predominantly White adolescents in Study 1 by examining reactivity to conflict in a racially and ethnically diverse sample of preschool children.

According to developmental models of interparental conflict (Davies et al., 2006; Kitzmann, Gaylord, Holt, & Kenny, 2003), preschool children adapt to interparental conflict in developmentally distinct ways based on a unique confluence of intraorganismic and family processes. Meta-analytic results have indicated that associations between children’s exposure to destructive interparental conflict and their psychological problems are particularly pronounced during the preschool period (Kitzmann et al., 2003). Moreover, children’s patterns of responding to interparental conflict are theorized to underpin their heightened sensitivity to interparental conflict during the preschool period. As children traverse through the preschool years, advances in social perspective taking and information processing abilities are posited to increase children’s concerns about the welfare of their parents during conflicts (Davies et al., 2006). Consistent with this finding, the limited research findings on involvement during early childhood indicate that intervention in parental and adult disputes increases significantly from toddlerhood to the late preschool years (Cummings, 1994; Cummings, Zahn-Waxler, & Radke-Yarrow, 1984). The limited sophistication and effectiveness of preschooler intervention efforts may also increase their vulnerability to subsequent psychological problems relative to school-aged children (Cummings et al., 1984). Moreover, in comparison to older children, heightened involvement is also commonly accompanied by preschooler experiences of greater fear, threat, and perceived coping inefficacy in response to interparental conflict (Cummings, Vogel, Cummings, & El-Sheikh, 1989; Grych, 1998; Jouriles, Spiller, Stephens, McDonald, & Swank, 2000).

At a conceptual level, our objective was to examine the replicability of the Study 1 findings. Consistent with Study 1, we examined whether children’s internalizing and externalizing symptoms were predicted in similar or distinct ways from the synergistic interaction between emotional reactivity and the two prevailing types of involvement in the literature: (a) mediation, consisting of comforting, distracting, solving the problem, or directly interceding in the conflict; and (b) coercive control, characterized by bossy, angry, and aggressive strategies for altering the course of the conflict. However, as a further test of the specificity and generalizability, we also expanded our multi-dimensional assessment of involvement to examine appeasing forms of intervening in the conflict. Appeasing involvement is specifically characterized by efforts to placate parents through inauthentic, demonstrative displays of charm, positivity, and over-bright behavior (e.g., Emery, 1982; Garcia O’Hearn, Margolin, & John, 1997; Johnston, et al., 2009). Although research has yet to systematically examine the developmental implications of appeasement, different sources of evidence highlight its prevalence in the face of interparental conflict. Practitioners have repeatedly emphasized that responding to interparental conflict with facades of charm and positivity is relatively common in childhood (Emery, 1982; Johnston et al., 2009). Moreover, as an indicator of appeasement, maternal diary reports indicated 15% of children exhibited “unusually well-behaved” responses to interparental conflict during a 5-week period, a percentage that was relatively similar to indicators of coercive (e.g., 14% “misbehaved”; 19% “took sides”) involvement (Garcia O’Hearn et al., 1997). Thus, as a first foray into examining appeasement in models of children’s psychological problems, we tested its role as a predictor and moderator in pathways between children’s emotional reactivity and their psychological difficulties.

Finally, although parental reports of children’s overall levels of involvement in interparental conflict have been shown to be psychometrically sound, parents’ tendencies to direct attention towards their partners during conflicts likely undermines their ability to detect specific displays of emotional reactivity (e.g., veiled expressions of fearful distress) and subtle forms of involvement (e.g., appeasing behaviors). Study 2 was designed to address this barrier through the implementation of an observational coding system that was designed to sensitively identify children’s emotional reactivity and forms of involvement in the context of an actual interparental disagreement. Moreover, we strengthened the multi-informant measurement approach in Study 1 by incorporating multiple informants and methods into the assessment battery of Study 2. In comparison to the prospective design of Study 1, our analysis of the interactions between emotional reactivity and the forms of involvement as predictors of subsequent change in children’s psychological adjustment also took place over a shorter, one-year span. Our selection of narrower developmental interval in Study 2 was guided by conceptualizations of early childhood as a period of considerable malleability in functioning and our goal of minimizing the possibility of missing significant change in children’s adjustment (e.g., Frankenhuis & Del Giuidice, 2012; Shaw, Bell, & Gilliom, 2000).

Methods

Participants

Participants included 243 families (i.e., mother, intimate partner, and preschool child) recruited through multiple agencies in a moderate-sized metropolitan area in the Northeast. To obtain a sample from diverse demographic backgrounds, our specific recruitment streams included, but were not limited to: local preschools, Head Start agencies, Women, Infants, and Children (WIC) programs, public and private daycare providers, and internet sites serving children and families from a variety of racial and ethnic backgrounds. The average age of children at Wave 1 was 4.60 years (SD = .44), with 56% of the sample consisting of girls. The longitudinal design consisted of two annual measurement occasions beginning when children were in their last year of preschool. The retention rate across the waves was 97%.

Median household income of the families was $36,000 per year (range = $2,000 – $121,000), with most families (69%) receiving public assistance. Approximately 19% of the parents did not earn a high school diploma or GED, with the median education for the sample consisting of a GED or high school diploma. Almost half of the families were Black or African American (48%), followed by smaller percentages of families who identified as White (43%), multi-racial (6%), or another race (3%). Approximately 16% of the family members were Latino. At Wave 1, 99% of the mothers and 74% of their partners were biological parents. Parents lived together an average of 3.36 years and had, on average, daily contact with each other and the child (range = daily to two or three days a week). Approximately half of the adults (47%) were married. Prevalence rates of interparental conflict and aggression in the sample were comparable to other estimates in community and national surveys. For example, mothers and fathers reports of annual rates of interparental violence on the Conflict Tactics Scale (CTS-2; Straus et al., 1996) were between 13% and 17%; a level similar to estimates (i.e., 16%) in national and community samples (El-Sheikh et al., 2001; Straus et al., 1996).

Procedures and Measures

Parents and children visited our research center laboratory at each of two annual waves of data collection. All research procedures were approved by the Institutional Review Board prior to conducting the study. Families were compensated monetarily for their participation.

Children’s responses to interparental conflict

To obtain observational assessments of children’s reactivity to interparental conflict at Wave 1, mothers and their partners participated in an interparental interaction task in which they discussed common, problematic disagreements. Following other interparental interaction tasks (Grych, 2002; Gordis, Margolin, & John, 2001), parents were informed during consent and prior to the interaction that their children would join them in the room as they discuss the issues. While the child was in a separate room, parents first selected problematic issues to discuss during the ten-minute task. After parents selected disagreement issues that they were comfortable discussing, an experimenter escorted the child into the room and introduced them to a set of toys. The parents then engaged in the interaction after the experimenter left the room. The task was video-recorded for subsequent coding.

Trained raters coded the video records of children’s reactivity to interparental conflict along molar scales of fearful distress and the three forms of involvement, including mediation, appeasement, and coerciveness. Each molar scale ranged from 1 (Not at all characteristic) to 9 (Mainly characteristic). As the measure of emotional reactivity, fearful distress was defined as children’s displays of anxiety, tension, fear, worry, vigilance, or emotional upset through facial (e.g., open mouth staring wide-eyed), postural (e.g., freezing), or gestural (e.g., wringing hands) expressions. Coerciveness was characterized by aversive, bossy, and controlling behaviors that functioned to regulate family activities in a way that undermined parental authority. Whereas mild forms of coercive control may have involved whining and complaining, more significant levels consisted of bossy, angry, and domineering approaches to interrupting parental activities (e.g., yelling “stop”; name-calling; triangulation by directly siding with one parent and challenging or arguing with the other parent). Mediation, by contrast, was reflected in forms of intervention that functioned to regulate interparental interactions without usurping the authority of the parents. In milder forms, mediation commonly involved some repeated, but wellregulated, and assertive efforts to interrupt the parents without displays of irritation or protesting (e.g., asking for help with toys). At more extreme levels requiring considerable forethought and risk, mediation was characterized by concerted efforts to comfort or help the parents, serve as a mediator (e.g., requests or pleading for the parents to stop bickering or get along), or very subtle forms of triangulation that were unlikely to be identified by the alienated parent as an alliance against him or her (e.g., subtly but selectively providing comfort to one parent). As the final form of involvement, appeasement consisted of children’s attempts to placate the parents through coy, ingratiating, and inauthentic, overenthusiastic behaviors that masked underlying anxiety, apprehension, and awkwardness (e.g., sudden, unexplainable intense smiling; reverting to “baby talk”). Two trained coders independently rated 21% of the videos to assess interrater reliability. Intraclass correlation coefficients ranged from .91 to .95.

Children’s internalizing symptoms

Mothers participated in a clinical diagnostic interview, the Diagnostic Interview Schedule for Young Children, Version IV (DISC-IV-YC; Luby et al., 2002; Luby, Mrakotsky, Heffelfinger, Brown, & Spitnagel, 2004). The DISC-IV-YC is a structured psychiatric interview designed for administration by lay interviewers with minimal training. The DISC-IV-YC yields psychometrically sound, dimensional ratings of psychopathology symptoms. Accordingly, to obtain latent constructs of children’s internalizing symptoms at each wave, we used three dimensional assessments based on symptom tallies (1 = present; 0 = absent) from DISC-IV-YC modules. As the first assessment, the Major Depressive Disorder module consisted of symptom counts from 22 items (e.g., “Nothing fun / disinterested in anything/sad”; α = .74 at Wave 1 and α = .75. at Wave 2). For the second measure, the Generalized Anxiety Disorder module was comprised of 12 symptoms (e.g., “Worried a lot about small mistakes made on tasks or activities”; α = .67 at Wave 1 and α = .69 at Wave 2). As the final assessment, we utilized the Separation Anxiety Disorder module (13 symptoms; e.g., “Afraid being left without family”; α = .65 at each Wave).

Children’s externalizing problems

Three measures were used as indicators of a latent construct of children’s externalizing symptoms at each time point. The first two indicators were derived from symptom tallies (1 = present; 0 = absent) from the Oppositional Defiant Disorder (ODD) and Attention Deficit Hyperactivity Disorder (ADHD) modules of the DISC-IV-YC psychiatric interview with the mother. The ODD scale contained 12 items (e.g., “Done things just to annoy people/make them mad”), and the ADHD scale consisted of 23 items (e.g., “Often had trouble waiting for turns [as in standing in line]”). Alpha coefficients were .87 and .83 for the ODD scale at Waves 1 and 2, respectively, and .87 at Wave 1 and .90 at Wave 2 for the ADHD scale. As the final indicator, mothers completed the externalizing scale from the MacArthur Health and Behavior Questionnaire (HBQ; Ablow et al., 1999). The externalizing scale is comprised of the sum of the 31 items from Oppositional Defiant (e.g., “Has temper tantrums or hot temper”), Conduct Problems (“Lies or cheats”), Overt Hostility (“Kicks, bites, or hits other children”), and Relational Aggression (“Tries to get others to dislike a peer”) subscales. Responses alternatives for each scale were: 0 (Never or not true), 1 (Sometimes or somewhat true), and 2 (Often or very true). Internal consistencies for the scale were .90 at Wave 1 and .92 at Wave 2.

Covariates

Consistent with Study 1, two demographic covariates derived from an interview with the mother at Wave 1 included (a) child gender and (b) total annual household income. To control for the potential confounding effects of children’s exposure to destructive interparental conflict, two trained raters coded the video records for maternal and partner hostility in the interparental interaction task to generate an interparental conflict variable. The interparental conflict variable specifically consisted of coder ratings of maternal and partner Anger and Aggression on molar scales. Whereas the Anger scale was designed to assess facial expressions, verbalizations, and postural and gestural displays of irascibility, frustration, and irritation, the Aggression scale indexed verbalizations and behaviors that were intended to harm the partner either physically or psychologically (e.g., demeaning, insulting, name-calling, threatening, cruel remarks). Each molar scale ranged from 1 (Not at all characteristic) to 9 (Mainly characteristic). Interrater reliability coefficients, which were calculated based on two coders’ independent ratings of 20% of the interactions, ranged from .91 to .95 across maternal and partner anger and aggression codes. In accord with Study 1, the four observational ratings of destructive conflict were standardized and aggregated to form a single composite of interparental conflict (α = .73).

Results

Descriptive and Preliminary Analyses

For descriptive purposes, Table 2 provides the means, standard deviations, and correlations for primary variables in the study. Consistent with Study 1, we assessed change in internalizing and externalizing symptoms through the use of LDS modeling across the two waves using strong factorial variance procedures to maximize measurement equivalence across time (Widaman et al., 2010). Following the Study 1 procedures (see Figure 2 for a conceptual illustration), we first tested separate unconditional LDS models for internalizing and externalizing symptoms to determine whether there was sufficient variability in intraindividual changes in children’s psychological problems to proceed to our primary analyses (McArdle, 2009). Both models fit the data well: χ2 (9, N = 243) = 25.72, p = .23, RMSEA = .088, CFI = 0.97, and χ2/df ratio = 2.86 for internalizing symptoms; and χ2 (9, N = 243) = 11.35, p = .25, RMSEA = .033, CFI = 1.00, and χ2/df ratio = 1.26, for externalizing symptoms. Statistically significant variances of 9.19 (z = 4.72) for externalizing symptoms and 3.74 (z = 5.52) for internalizing symptoms indicated that there were significant individual differences in mean changes in children’s problems from Time 1 to Time 2. Therefore, we proceeded to our primary goal of examining children’s forms of involvement as a moderator of associations between their emotional reactivity and internalizing and externalizing symptoms.

Table 2.

Means, Standard Deviations, and Correlations for the Primary Variables in the Study 2 Analyses

Mean SD 1 2 3 4 5 6 7 8 9 10 11 12 13 14 15 16 17 18
Wave 1 Covariates
 1. Child Gender 1.44 0.50
 2. Household Income 41.18 23.20 .05
 3. Interparental Conflict 0.00 0.74 −.02 −.14*
Wave 1 Child Reactivity to Conflict
 4. Emotional Reactivity 2.89 1.62 −.06 −.08 .22*
 5. Mediation 3.56 2.02 .06 .14* −.11 −.17*
 6. Appeasing 3.11 1.94 −.13* . 13* .03 .02 .39*
 7. Coerciveness 2.48 2.27 −.06 −.08 −.11 −.10 .31* . 00
Wave 1 Internalizing Symptoms
 8. DISC Depression 3.71 3.05 .02 −.02 .05 .10 .00 . 07 .18*
 9. DISC Generalized Anxiety 1.76 1.92 −.08 −.10 .12 .19* −.10 . 10 −.01 .59*
 10. DISC Separation Anxiety 2.78 2.18 −.15* −.22* .16* .07 −.02 . 04 .11 .54* 46*
Wave 1 Externalizing Symptoms
 11. DISC ODD 5.28 3.04 .13* .19* .03 −.04 .20* .07 .15* .48* .26* .30*
 12. DISC ADHD 8.71 5.40 .09 −.14* .08 −.02 .02 −.02 .18* .51* .32* .40* .49*
 13. HBQ Externalizing 8.25 7.07 .08 −.03 .19* .07 −.06 −.05 .06 .31* .20* .24* .52* 49*
Wave 2 Internalizing Symptoms
 14. DISC Depression 3.18 2.93 .10 −.09 .10 .15* .10 −.01 .11 .50* .31* .37* .39* .48* .33*
 15 DISC Generalized Anxiety 1.89 2.04 .04 .02 .04 .19* −.01 .13 .10 .40* .49* .32* .25* .31* .15* .57*
 16. DISC Separation Anxiety 2.32 2.17 .01 −.19* .17* .11 .01 .03 .14* .34* .34* .51* .30* .39* .17* .53* .54*
Wave 2 Externalizing Symptoms
 17. DISC ODD 4.76 3.34 .07 .14* .08 .02 .14* .03 .12 .39* .13 .30* .63* .32* .43* .55* .35* .36*
 18. DISC ADHD 8.44 5.99 .18* −.19* .15* .05 −.03 −.06 .12 .48* .35* .40* .44* .74* .46* .62* .34* .42* .47*
 19. HBQ Externalizing 7.95 7.62 .08 −.11 .15* .05 −.05 .00 .09 .36* .18* .31* .47* .43* .76* .42* .20* .25* .54* .56*

Note. Child gender: 1 = female; 2 = male. p ≤ .05.

Analytic Plan

To test the moderator hypotheses, six analytic models were estimated based on successively coupling children’s emotional reactivity with each of the three forms of involvement in predicting LDS change for internalizing and externalizing symptoms separately (i.e., 1 predictor × 3 forms of involvement × 2 outcomes). Consistent with the unconditional LDS analyses, strong factorial invariance procedures were implemented in modeling change in child adjustment (i.e., the loadings and intercepts of the same indicators across time were constrained to be equal). As depicted in the conceptual illustration in Figure 6, emotional reactivity, involvement in the conflict (i.e., mediation, appeasement, or coerciveness), and their multiplicative product were examined as predictors of LDS change in children’s internalizing or externalizing symptoms while also specifying the Wave 1 form of child adjustment (i.e., autoregressive path), child gender, family income, and interparental conflict as covariates. As continuous predictors, the child reactivity variables (i.e., emotional reactivity, mediation, appeasement, and coerciveness) were centered to reduce multicollinearity in the moderator analyses. In accord with the analytic approach in Study 1, correlations were also specified between: (a) the primary predictors (i.e., emotional reactivity and the form of involvement), (b) the covariates, (c) the covariates and primary predictors, (d) the Wave 1 latent construct of children’s psychological functioning and each of the covariates and predictors, (e) the same measure of psychological difficulties across the two time points (see Figure 6). Structural equation models were estimated using full-information maximum likelihood (FIML) in Amos 22.0 to estimate missing data (i.e., data were missing for 2.1% of the values) and retain the full sample for primary analyses (Enders, 2001).

Figure 6.

Figure 6

A conceptual illustration of Study 2 LDS analyses of the interaction between children’s emotional reactivity and involvement in predicting children’s psychological symptoms.

Primary Analyses

Table 3 shows the standardized loadings of the manifest indicators onto their latent constructs for each of the six analytic models. In support of the measurement model, all loadings were significant (all ps < .001) and strong in magnitude, ranging from .67 to .78. Fit indices for the models were all satisfactory. For succinctness, we report the mean and range of key fit indices across the six analytic models: (a) mean χ2/df ratio = 2.24 (range = 2.02 to 2.43); (b) mean RMSEA = .072 (range .065 to .076); and (c) mean CFI = .94 (range = .92 to .96).

Table 3.

Standardized Loadings of the Indicators of the Latent Psychological Problems Variables in the Study 2 LDS Analyses

Loadings of the Indicators for Each Model

Dependent Measures Child Mediation Child Appeasing Child Coerciveness
Internalizing Symptoms Model
 W1 DISC Depression .77 .77 .78
 W1 DISC Generalized Anxiety .73 .73 .72
 W1 DISC Separation Anxiety .69 .69 .69
 W2 DISC Depression Symptoms .78 .78 .79
 W2 DISC Generalized Anxiety .71 .72 .71
 W2 DISC Separation Anxiety .71 .71 .70
Externalizing Symptoms Model
 W1 DISC ODD Symptoms .72 .71 .72
 W1 DISC ADHD .67 .68 .68
 W1 HBQ Externalizing Behavior .72 .73 .71
 W2 DISC ODD Symptoms .72 .70 .71
 W2 DISC ADHD .70 .72 .72
 W2 HBQ Externalizing Behavior .76 .77 .76
Externalizing symptoms

Results of the structural models depicting the interplay between children’s emotional reactivity and each of the forms of involvement are shown in the left panel of Table 4 under columns designated as Models 1a (mediation), 1b (appeasement), and 1c (coerciveness). None of the main effects involving emotional reactivity, forms of involvement, or covariates as predictors of LDS change in externalizing symptoms were significant. As hypothesized, the interaction between children’s emotional reactivity and mediation was significantly associated with increases in externalizing symptoms over the one-year period, β = .22, p < .01. The moderating effect was first clarified by graphically plotting and calculating simple slopes of emotional reactivity at high (+1 SD) and low (−1 SD) levels of children’s mediation in the parental conflict. Consistent with recommendations followed in Study 1 (Roisman et al., 2012), we conducted simple slope plots and calculations at −2 SD and + 2 SD from the centered mean of emotional reactivity. Findings from the simple slope analyses revealed that emotional reactivity predicted increases in children’s externalizing symptoms at high, b = 0.46, p < .05, but not low levels, b = −0.19, p = .22, of mediation. Likewise, the graphical plot depicted in Figure 7a revealed a disordinal interaction that is similar to the form of the Study 1 plot and the vulnerable-adaptive model.

Table 4.

Results of Standardized Structural Paths in the Study 2 LDS Analyses Examining the Interplay Between Emotional Reactivity and the Three Forms of Involvement

Δ Externalizing Symptoms Δ Internalizing Symptoms

Model 1a Model 1b Model 1c Model 2a Model 2b Model 2c
Covariates:
 Child Gender .08 .12 .11 .14 .15* .16*
 Interparental Conflict .05 .06 .06 .07 .08 .07
 Family Income −.16 −.16 −.15 −.03 .01 .01
 Autoregressive Path −.08 −.07 −.06 −.42* −.40* −.42*
Predictors:
 Emotional Reactivity .10 .04 .06 .16* .13 .18*
 Mediation .00 .11
 Emot. Reactivity × Mediation .22* .11
 Appeasing .04 .00
 Emot. Reactivity × Appeasing .00 .16*
 Coercive −.01 .12
 Emot. Reactivity × Coercive .06 .21*

Note. Columns with the “a”, “b”, and “c” suffixes depict the results of analytic models examining children’s mediation, appeasing behavior, and coercive control, respectively.

*

p < .05.

Figure 7.

Figure 7

Figure 7

Figure 7

a. A graphical plot of the interaction between children’s emotional reactivity and their mediation in predicting changes in their externalizing symptoms over a one-year period.

b. A graphical plot of the interaction between children’s emotional reactivity and their appeasement in predicting changes in their internalizing symptoms over a one-year period.

c. A graphical plot of the interaction between children’s emotional reactivity and their coerciveness in predicting changes in their internalizing symptoms over a one-year period.

To more definitely characterize the nature of the interaction and its similarity to the vulnerable-adaptive model, we calculated the same quantitative indices used in Study 1. First, the RoS on X test was conducted to examine whether mediation was significantly associated with externalizing symptoms at high (+ 2 SD) and low (− 2 SD) levels of emotional reactivity. The results indicated that children’s mediation in interparental conflict predicted higher externalizing symptoms at high levels (+2 SD) of emotional reactivity, b = 0.53, p < .05. Conversely, higher mediation was associated with decreases in externalizing symptoms when children exhibited low levels (−2 SD) of emotional reactivity, b = −0.51, p < .05. As a further index of the relative viability of each of the three moderator models in Figure 1, we calculated the PoI interaction index, or ratio of the improved outcomes of children evidencing high involvement relative to the aggregate of both improved and impaired outcomes. In accord with the findings from Study 1, the resulting PoI value of .49 fell within the range of strong support (i.e., .40 to .60) for the vulnerable-adaptive model.

Internalizing symptoms

The right panel of Table 4 under the Models 2a (mediation), 2b (appeasement), and 2c (coerciveness) displays the moderator results of emotional reactivity and each form of involvement in predicting children’s internalizing symptoms. Consistent with prior associations between initial level and change in psychological functioning (King, King, McArdle, Shalev, & Doron-LaMarca, 2009), the specification of the autoregressive path revealed that Wave 1 levels of internalizing symptoms were negatively correlated with its subsequent change over time in each of the models, with βs ranging from −.40 to −.42, all ps < .001. For the appeasement and coerciveness analyses, significant paths between child gender and change in internalizing symptoms (βs = .15 and .16, ps < .05) revealed that girls evidenced greater increases in internalizing symptoms than did boys. Finally, inspection of children’s responses to interparental conflict as predictors (i.e., the Predictors section of Table 4) showed that emotional reactivity predicted greater internalizing symptoms over time for the mediation, β = .16, p < .05, and coerciveness, β = .18, p < .05, models. Of greater relevance to the aims of the study, the interaction between emotional reactivity and involvement significantly predicted latent changes in children’s internalizing symptoms for appeasement (Model 2b), β = .16, p < .05, and coerciveness (Model 2c), β = .21, p < .01 models, but not the mediation (Model 2a) analysis. The graphical plot for the moderating role of appeasement is shown in Figure 7b. Simple slope analyses revealed that children’s emotional reactivity predicted increases in their internalizing symptoms at high, b = 0.35, p < .01, but not low levels, b = −0.04, p = .73, of appeasement. Results of the RoS on X and PoI tests for appeasement revealed a similar pattern to the moderating findings for mediation in predicting children’s externalizing symptoms. Appeasing behavior was associated with increases in children’s internalizing symptoms at high levels (+2 SD) of emotional reactivity, b = 0.33, p < .05. In contrast, appeasing behavior predicted significant decreases in internalizing symptoms over time when children exhibited low levels (−2 SD) of emotional reactivity, b = −0.33, p < .05. Likewise, the PoI coefficient of .50 revealed strong support for the vulnerable-adaptive model and its hypothesis that there is a comparable balance of improved and impaired outcomes for high involvement across the range of the emotional reactivity variable.

Figure 7c shows the graphical plot for the moderating effects of coerciveness. In the simple slope analyses, emotional reactivity predicted increases in children’s internalizing symptoms over the one-year period only at high levels of coerciveness, b = 0.49, p < .001. Emotional reactivity failed to predict children’s internalizing symptoms at low levels of coerciveness, b = −0.07, p = .60. In accord with the other moderator results, the RoS on X test further revealed that coercive control predicted increases in internalizing symptoms when children experienced heightened (+ 2 SD) emotional reactivity, b = 0.51, p < .01. Conversely, at low levels of emotional reactivity, children’s coercive control was associated with lower levels of internalizing symptoms over time, b = −0.30, p < .05. Finally, although the PoI index of .26 was lower than the other interactions, it still fell well above the cutoff (.16) for providing support for the vulnerable-adaptive over vulnerable-reactive model.

General Discussion

Children’s involvement has been conceptualized as a pathogenic process in several theories of interparental conflict (Davies & Cummings, 1994; Emery, 1989; Grych & Fincham, 1990). However, research on involvement as a predictor of child psychopathology has yielded inconsistent findings even when it is operationalized in EST as a sign of insecurity (e.g., Buehler et al., 2007). Increasing emphasis on identifying higher-order patterns of children’s reactivity in EST raises the possibility that involvement primarily serves as a moderator of other signs of emotional security (Davies & Martin, 2013). Thus, as the central mechanism underlying the organization of the emotional security system, we examined whether the role of children’s emotional reactivity to interparental conflict as a predictor of their internalizing and externalizing symptoms varied as a function of their involvement in the conflicts. Against a research landscape that has yet to test the developmental implications of the synergistic interplay between signs of children’s insecurity in the interparental relationship, the findings of this study were novel in demonstrating that the interaction between children’s emotional reactivity and involvement predicted subsequent increases in children’s psychological symptoms. Across the two studies, vulnerability to adjustment problems was disproportionately greater for children who exhibited high levels of emotional reactivity in conjunction with heightened involvement in interparental conflicts. Less consistency, however, was evident if the moderating effects of the forms of involvement are interpreted in relation to children’s specific forms of maladjustment. Thus, although prospective associations between children’s emotional reactivity and their externalizing symptoms varied significantly as a function of their mediation in conflicts across both studies, children’s coercive involvement varied across the two studies in whether its role as a moderator of emotional reactivity was applicable in predicting their externalizing symptoms (Study 1) and internalizing symptoms (Study 2).

Consistent with previous research (e.g., Buehler et al., 2007; Davies & Cummings, 1998), findings from both studies indicated that emotional reactivity to conflict predicted increases in child psychopathology (i.e., externalizing problems for adolescents in Study 1; internalizing symptoms for children in Study 2). However, the main effects in the analyses were regularly qualified by interactions between children’s emotional reactivity and involvement. In dissecting the interactions, findings across the two studies consistently revealed that children’s emotional reactivity predicted subsequent psychological problems only when they exhibited heightened involvement in parental conflicts. More specifically, the selectivity of emotional reactivity as a risk was evidenced at high levels of multiple forms of involvement, including mediation, appeasement, and coercive control.

The findings beg the question of why different forms of involvement magnified the negative sequelae of children’s emotional reactivity to interparental conflict. On the one hand, it is possible that common processes underlie the moderating role of each type of involvement. For example, regardless of whether involvement is expressed specifically through directly managing (i.e., mediation), dominating (i.e., coercive), or indirectly placating (i.e., appeasing behavior) upset parents, the different types of intervention may signify common underlying enmeshment in adverse family events (Davies & Forman, 2002). If the dimensions of involvement each reflect high investment in parental quarrels, then it follows that fear, worries, and distress in high conflict homes may only become psychologically burdensome for children when they are also regularly entangled in adult problems. On the other hand, different forms of involvement may also give rise to similar moderating effects through the operation of different mechanisms. For example, by virtue of its more potent aversive qualities relative to mediating and appeasing behaviors, coercive methods of managing interparental conflicts may evoke more negativity from parents, place strain on their relationship with the children, and ultimately undermine the quality of their own romantic relationship (e.g., Schermerhorn et al., 2007). Resulting increases in children’s exposure to family discord, in turn, may catalyze the risk posed by children’s emotional reactivity over time. In accord with this possibility, research has shown that children’s emotional insecurity in the interparental relationship is a stronger predictor of their psychological problems when they experience more parenting difficulties, parent-child relationship difficulties, and family disengagement (Davies, Harold, Goeke-Morey, & Cummings, 2002).

However, exclusively interpreting the effects of involvement in altering associations between emotional reactivity and children’s psychopathology does not permit a comprehensive characterization of its moderating role. More specifically, it does not afford an authoritative analysis of whether the potentiating role of involvement is vulnerable-reactive or vulnerable-adaptive in form. Although both models propose that emotional reactivity is a stronger predictor of children’s psychological problems when involvement is high, they differ in their conceptualization of whether involvement confers any conditional advantages for children. The vulnerable-reactive model proposes that involvement is generally maladaptive regardless of the degree of emotional reactivity experienced by children (see Figure 1b). Although the vulnerable-adaptive model shares the hypothesis that involvement may be a risk factor when emotional reactivity is high, it is distinctive in theorizing that high levels of involvement may decrease children’s vulnerability for psychopathology when emotional reactivity is low (see Figure 1c). Follow-up analyses designed to directly test the comparative fit of the two complementary predictions consistently supported the vulnerable-adaptive model over the vulnerable-reactive model. Across all five significant interactions, high involvement was a significant predictor of increases in psychological problems when children experienced high levels of emotional reactivity, but also of decreases in psychological problems when they exhibited low levels of emotional reactivity. Moreover, the PoI index indicated that the proportion of the interaction that reflected advantageous psychological outcomes fell between .24 and .54 (M = .44), values that were well within the range for supporting the hypothesis that involvement has adaptive and maladaptive implications depending on the level of emotional reactivity.

Our findings are consistent with the new pattern-based formulation of EST (Davies & Martin, 2013). According to this framework, involvement in the context of high levels of emotional reactivity may be characteristic of a “mobilizing” profile of vigorously defending against threat posed by interparental conflict while also proactively maintaining ties with the parents. A primary prediction is that this mobilizing profile may increase both internalizing and externalizing symptoms over time by engendering hypervigilance to social stimuli, self-consciousness, proclivity to experience shame, and impulsive and attention-seeking forms of risk-taking. Supporting this hypothesis, the results collectively indicated that heightened emotional reactivity and involvement predicted subsequent increases in internalizing symptoms in some analyses and greater externalizing symptoms in other analyses. In contrast, the pattern-based version of EST also proposes that involvement coupled with low levels of emotional reactivity may represent a broader “secure” pattern of responding indicative of well-regulated empathetic concern for the parents. Thus, when involvement is part of a broader pattern of security (i.e., low emotional reactivity), it may reduce children’s psychological problems by facilitating the enactment of: (a) affiliative goals and the accompanying acquisition of social skills and (b) caregiving goals and their cultivation of empathy, sympathy, and helping behavior.

The vulnerable-adaptive moderating results for involvement may also inform distinctions drawn between adaptive and destructive parentification in family systems conceptualizations (e.g., Byng-Hall, 2002; Jurkovic et al., 1999). In these frameworks, adaptive parentification is differentiated from destructive parentification based on an analysis of whether the quality (e.g., brief, developmentally appropriate) of children’s involvement in family affairs preserves or promotes children’s autonomy. Our findings indicate that emotional reactivity may be a key source for discriminating between these two forms of parentification. Consistent with this interpretation, Byng-Hall (2002) proposed that destructive parentification results from children who organize their distress and fear of their caregivers into strategies designed to control parental behavior. In contrast, involvement under conditions of low fearful distress (i.e., emotional reactivity) may signify self-confidence and self-efficacy that ultimately promotes the development of competence and identity.

Discussion of the limitations of the study is also necessary in fully interpreting the findings. Although confidence in our conclusions are bolstered by the replication of findings across studies and different forms of involvement, the moderating findings for involvement do not distinguish between internalizing and externalizing symptoms in readily interpretable ways. For example, based on empirically documented links between aggressive forms of involvement in family conflict and children’s externalizing symptoms (e.g., Davis et al., 1998), it might be expected that coercive ways of intervening in interparental conflicts would have increased children’s externalizing symptoms when emotional reactivity was high. However, our results from Study 2 revealed that the interplay between coerciveness and emotional reactivity specifically predicted increases in children’s internalizing symptoms. Likewise, appeasing behaviors in interpersonal contexts that are likely to evoke high levels of emotional reactivity (i.e., threatening, unsupportive) have been hypothesized to specifically increase internalizing symptoms (e.g., Gilbert, 2001). In contrast, our findings indicated that children who exhibited high levels of emotional reactivity and appeasing forms of involvement were at disproportionately higher risk for developing externalizing symptoms over time. Our analyses did not permit direct tests of whether the moderating effects of the various forms of involvement differed significantly from each other in the prediction of children’s internalizing and externalizing problems. Thus, without further research, it would be premature to draw any conclusions about the existence of differential pathways between emotional reactivity and the different forms of involvement and specific types of psychological problems.

Moreover, although our null findings on associations between forms of involvement and psychological problems correspond with empirical inconsistencies in the broader literature, it is important to note that other conceptual and methodological approaches to capturing involvement may increase its power as a predictor of children’s adjustment. First, different ways of operationalizing involvement might produce different patterns of results. For example, individual differences in the level of planfulness and sophistication in involvement strategies may be meaningfully associated with different child adjustment outcomes (Cummings et al., 1991). Second, it is possible that different design and analytic approaches to assessing prospective associations between forms of reactivity to interparental conflict and children’s adjustment might yield stronger effects for involvement. For example, multilevel modeling analyses revealed that intraindividual increases in involvement over time were associated with corresponding rises in children’s externalizing problems (Jouriles et al., 2014). Third, although our findings are bolstered by replication across our samples of families who varied widely in demographic backgrounds, caution should be exercised in translating these findings to high-risk or clinical samples of children. Fourth, our approach does not rule out the possibility that the interplay between emotional reactivity and forms of involvement in interparental conflict contexts is a proxy for children’s temperament or their broader styles of coping with a wide array of stressful situations. By the same token, prior research has shown that emotional reactivity and involvement in contexts of interparental conflict are distinct from broader patterns of psychological functioning in their composition and power to predict children’s mental health outcomes (e.g., Cummings, George, McCoy, & Davies, 2012; Davies, Manning, & Cicchetti, 2013; Davies, Martin, & Cicchetti, 2012). In addition, analyses between these forms of reactivity to interparental conflict and broader indices of emotional and behavioral reactivity to stimuli from the two datasets in this paper generally yielded weak to modest correlations, thereby providing further support for the unique nature of emotional reactivity and involvement as constructs. (Results of correlational analyses from the two studies are available from the authors upon request).

Finally, our goal of exploring children’s involvement in conflict as a moderator of pathways between their emotional reactivity and adjustment problems within two age periods does not definitively address some important developmental questions. At this early stage of research, little is known about whether the synergistic interaction between emotional reactivity and involvement is associated with outcomes in childhood and adolescence through different or similar mechanisms. On the one hand, coercive forms of involvement in conjunction with emotional reactivity may increase vulnerabilities through common pathways in childhood and adolescence. For example, as part of a coercive transactional process, negative reactivity to interparental conflict by children and adolescents is proposed to ultimately increase their psychological problems by evoking difficulties between parents over time (e.g., Schermerhorn et al., 2010). On the other hand, adolescents may share similar outcomes with children through different configurations of protective and risk mechanisms. For example, the likelihood of experiencing psychological problems in the wake of high emotional reactivity and involvement (e.g., mediation) may increase in adolescence by virtue of their greater sensitivity to detect and comprehend the negative repercussions of interparental difficulties for the family (Davies, Myers, Cummings, & Heindel, 1999). By the same token, these heightened developmental vulnerabilities may be counteracted by the greater abilities of teens to actively regulate distress through cognitively structuring and distancing themselves from interparental difficulties (Sandler, Tein, & West, 1994). Therefore, more research is needed to systematically explore the differences and commonalities in the mechanisms and sequelae of involvement across developmental periods.

In conclusion, our findings incrementally advance the knowledge on children’s coping with interparental conflict by offering the first test of the multiplicative interplay between emotional reactivity and forms of involvement as predictors of their psychological maladjustment. Building on previous empirical documentation of involvement as a weak to modest predictor of children’s psychopathology, our findings indicated that involvement functioned more consistently as a moderator of children’s emotional reactions to interparental conflict. Across both studies, prospective associations between emotional reactivity to interparental conflict and psychological symptoms were more pronounced for children who exhibited high levels of involvement in their parents’ disagreements. In addition, the developmental benefits and costs of involvement varied as a function of emotional reactivity. Consistent with the pattern-based formulation of EST and family systems models (e.g., Byng-Hall, 2002; Davies & Martin, 2013), involvement in interparental conflict predicted increases in psychological problems for children who exhibited high emotional reactivity and decreases in psychological problems when children experienced low levels of emotional reactivity.

Acknowledgments

Study 1 was supported by the National Institute of Mental Health (R01 MH57318) awarded to Patrick T. Davies and E. Mark Cummings. Study 2 was supported by the Eunice Shriver Kennedy National Institute of Child Health and Human Development (R01 HD065425) awarded to Patrick T. Davies and Melissa L. Sturge-Apple. Study 2 was conducted at Mt. Hope Family Center, University of Rochester. The authors are grateful to the children, parents, and community agencies who participated in these projects. We would also like to thank Mike Ripple, the Mt. Hope Family Center Staff, and the personnel at the Universities of Rochester and Notre Dame who assisted on these projects.

Contributor Information

Patrick T. Davies, Department of Clinical and Social Sciences in Psychology, University of Rochester

Jesse L. Coe, Department of Clinical and Social Sciences in Psychology, University of Rochester

Meredith J. Martin, Department of Clinical and Social Sciences in Psychology, University of Rochester

Melissa L. Sturge-Apple, Department of Clinical and Social Sciences in Psychology, University of Rochester

E. Mark Cummings, Department of Psychology, University of Notre Dame.

References

  1. Ablow J, Measelle JR, Kraemer HC, Harrington R, Luby J, Smider N, et al. The MacArthur Three-City Outcome Study: Evaluating multi-informant measures of young children’s symptomatology. Journal of the American Academy of Child and Adolescent Psychiatry. 1999;38:1580–1590. doi: 10.1097/00004583-199912000-00020. [DOI] [PubMed] [Google Scholar]
  2. Achenbach TM, Dumenci L, Rescorla LA. DSM-oriented and empirically based approaches to constructive scales from the same item pools. Journal of Clinical Child and Adolescent Psychology. 2003;32:328–340. doi: 10.1023/A:1021700430364. [DOI] [PubMed] [Google Scholar]
  3. Belsky J, Bakermans-Kranenburg MJ, van IJzendoorn MH. For better and for worse: Differential susceptibility to environmental influences. Current Directions in Psychological Science. 2007;16:300–304. doi: 10.1111/j.1467-8721.2007.00525.x. [DOI] [Google Scholar]
  4. Belsky J, Pluess M. Beyond diathesis–stress: Differential susceptibility to environmental influences. Psychological Bulletin. 2009;135:885–908. doi: 10.1037/a0017376. [DOI] [PubMed] [Google Scholar]
  5. Buchanan CM, Maccoby EE, Dornbusch SM. Caught between parents: Adolescents’ experience in divorced homes. Child Development. 1991;62:1008–1029. doi: 10.1111/j.1467-8624.1991.tb01586.x. [DOI] [PubMed] [Google Scholar]
  6. Buehler C, Lange G, Franck KL. Adolescents’ cognitive and emotional responses to marital hostility. Child Development. 2007;78:775–789. doi: 10.1111/j.1467-8624.2007.01032.x. [DOI] [PubMed] [Google Scholar]
  7. Byng-Hall J. Relieving parentified children’s burdens in families with insecure attachment patterns. Family Process. 2002;41:375–388. doi: 10.1111/j.1545-5300.2002.41307. [DOI] [PubMed] [Google Scholar]
  8. Covell K, Miles B. Children’s beliefs about strategies to reduce parental anger. Child Development. 1992;63:381–390. doi: 10.1111/j.1467-8624.1992.tb01634.x. [DOI] [PubMed] [Google Scholar]
  9. Crockenberg S, Langrock A. The role of specific emotions in children’s responses to interparental conflict: A test of the model. Journal of Family Psychology. 2001;15:163–182. doi: 10.1037/0893-3200.15.2.163. [DOI] [PubMed] [Google Scholar]
  10. Crockenberg SC, Leerkes EM, Lekka SK. Pathways from marital aggression to infant emotion regulation: The development of withdrawal in infancy. Infant Behavior and Development. 2007;30:97–113. doi: 10.1016/j.infbeh.2006.11.009. [DOI] [PubMed] [Google Scholar]
  11. Cummings EM. Marital conflict and children’s functioning. Social Development. 1994;3:16–36. doi: 10.1111/j.1467-9507.1994.tb00021.x. [DOI] [Google Scholar]
  12. Cummings EM, Ballard M, El-Sheikh M, Lake M. Resolution and children’s responses to interadult anger. Developmental Psychology. 1991;27:462–470. doi: 10.1037//0012-1649.27.3.462. [DOI] [Google Scholar]
  13. Cummings EM, Davies PT. Emotional security as a regulatory process in normal development and the development of psychopathology. Development and Psychopathology. 1996;8:123–139. doi: 10.1017/S0954579400007008. [DOI] [Google Scholar]
  14. Cummings EM, George MRW, McCoy KP, Davies PT. Interparental conflict in kindergarten and adolescent adjustment: Prospective investigation of emotional security as an explanatory mechanism. Child Development. 2012;83:1703–1715. doi: 10.1111/j.1467-8624.2012.01807.x. [DOI] [PMC free article] [PubMed] [Google Scholar]
  15. Cummings EM, Schermerhorn AC, Davies PT, Goekey-Morey MC, Cummings JS. Interparental discord and child adjustment: Prospective investigations of emotional security as an explanatory mechanism. Child Development. 2006;77:132–152. doi: 10.1111/j.1467-8624.2006.00861.x. [DOI] [PubMed] [Google Scholar]
  16. Cummings EM, Vogel D, Cummings JS, El-Sheikh M. Children’s responses to different forms of expression of anger between adults. Child Development. 1989;60:1392–1404. doi: 10.2307/1130929. [DOI] [PubMed] [Google Scholar]
  17. Cummings EM, Zahn-Waxler C, Radke-Yarrow M. Developmental changes in children’s reactions to anger in the home. Journal of Child Psychology and Psychiatry. 1984;25:63–74. doi: 10.1111/j.1469-7610.1984.tb01719.x. [DOI] [PubMed] [Google Scholar]
  18. Davies PT. Conceptual links between Byng-Hall’s Theory of Parentification and the Emotional Security Hypothesis. Family Process. 2002;41:551–555. doi: 10.1111/j.1545-5300.2002.41317.x. [DOI] [PubMed] [Google Scholar]
  19. Davies PT, Cicchetti D, Martin MJ. Towards greater specificity in identifying associations among interparental aggression, child emotional reactivity to conflict, and child problems. Child Development. 2012;83:1789–1804. doi: 10.1111/j.1467-8624.2012.01804.x. [DOI] [PMC free article] [PubMed] [Google Scholar]
  20. Davies PT, Cummings EM. Marital conflict and child adjustment: An emotional security hypothesis. Psychological Bulletin. 1994;116:387–411. doi: 10.1037/0033-2909.116.3.387. [DOI] [PubMed] [Google Scholar]
  21. Davies PT, Cummings EM. Exploring children’s emotional security as a mediator of the link between marital relations and child adjustment. Child Development. 1998;69:124–139. doi: 10.1111/j.1467-8624.1998.tb06138.x. [DOI] [PubMed] [Google Scholar]
  22. Davies PT, Cummings EM. Interparental discord, family process, and developmental psychopathology. In: Cicchetti D, Cohen DJ, editors. Developmental Psychopathology, Vol3: Risk, disorder, and adaptation. 2. Hoboken, NJ, US: John Wiley & Sons Inc; 2006. pp. 86–128. [Google Scholar]
  23. Davies PT, Forman EM. Children’s patterns of preserving emotional security in the interparental subsystem. Child Development. 2002;73:1880–1903. doi: 10.1111/1467-8624.t01-1-00512. [DOI] [PubMed] [Google Scholar]
  24. Davies PT, Forman EM, Rasi JA, Stevens KI. Assessing children’s emotional security in the interparental subsystem: The Security in the Interparental Subsystem (SIS) scales. Child Development. 2002;73:544–562. doi: 10.1111/1467-8624.00423. [DOI] [PubMed] [Google Scholar]
  25. Davies PT, Harold GT, Goeke-Morey M, Cummings EM. Children’s emotional security and interparental conflict. Monographs of the Society for Research in Child Development. 2002;67:1–129. doi: 10.1111/1540-5834.00205. [DOI] [PubMed] [Google Scholar]
  26. Davies PT, Manning LG, Cicchetti DD. Tracing the developmental cascade of children’s insecurity in the interparental relationship: The role of stage-salient tasks. Child Development. 2013;84:297–312. doi: 10.1111/j.1467-8624.2012.01844.x. [DOI] [PMC free article] [PubMed] [Google Scholar]
  27. Davies PT, Martin MJ, Cicchetti DD. Delineating the sequelae of destructive and constructive interparental conflict for children within an evolutionary framework. Developmental Psychology. 2012;48:939–955. doi: 10.1037/a0025899. [DOI] [PMC free article] [PubMed] [Google Scholar]
  28. Davies PT, Martin MJ. The reformulation of emotional security theory: The role of children’s social defense in developmental psychopathology. Development and Psychopathology. 2013;25:1435–1454. doi: 10.1017/S09545794130000709. [DOI] [PMC free article] [PubMed] [Google Scholar]
  29. Davies PT, Myers RL, Cummings EM. Responses of children and adolescents to marital conflict scenarios as a function of the emotionality of conflict endings. Merrill-Palmer Quarterly. 1996;42:1–21. [Google Scholar]
  30. Davies PT, Myers RL, Cummings EM, Heindel S. Adult conflict history and children’s responses to conflict: An experimental test. Journal of Family Psychology. 1999;13:610–628. [Google Scholar]
  31. Davies PT, Sturge-Apple ML, Bascoe SM, Cummings EM. The legacy of early insecurity histories in shaping adolescent adaptation to interparental conflict. Child Development. 2014;85:338–354. doi: 10.1111/cdev.12119. [DOI] [PMC free article] [PubMed] [Google Scholar]
  32. Davies PT, Winter MA, Cicchetti D. The implications of emotional security theory for understanding and treating childhood psychopathology. Development and Psychopathology. 2006;18:707–735. doi: 10.1017/S0954579406060354. [DOI] [PubMed] [Google Scholar]
  33. Davis BT, Hops H, Alpert A, Sheeber L. Child responses to parental conflict and their effect on adjustment. Journal of Family Psychology. 1998;12:163–177. doi: 10.1037/0893-3200.12.2.163. [DOI] [Google Scholar]
  34. Dearing E, Hamilton LC. Contemporary advances and classic advice for analyzing mediating and moderating variables. Monographs of the Society for Research in Child Development. 2006;71:88–104. doi: 10.1111/j.1540-5834.2006.00406.x. [DOI] [Google Scholar]
  35. El-Sheikh M, Harger J, Whitson SM. Exposure to interparental conflict and children’s adjustment and physical health: The moderating role of vagal tone. Child Development. 2001;72:1617–1636. doi: 10.1111/1467-8624.00369. [DOI] [PubMed] [Google Scholar]
  36. Emery RE. Family Violence. American Psychologist. 1989;44:321–328. doi: 10.1037/0003-066X.44.2.321. [DOI] [PubMed] [Google Scholar]
  37. Enders CK. A primer on maximum likelihood algorithms available for use with missing data. Structural Equation Modeling. 2001;8:128–141. doi: 10.1207/S15328007SEM0801_7. [DOI] [Google Scholar]
  38. Fosco GM, Grych JH. Emotional expression in the family as a context for children’s appraisals of interparental conflict. Journal of Family Psychology. 2007;21:248–258. doi: 10.1037/0893-3200.21.2.248. [DOI] [PubMed] [Google Scholar]
  39. Frankenhuis WE, Del Giudice M. When do adaptive developmental mechanisms yield maladaptive outcomes? Developmental Psychology. 2012;48:628–642. doi: 10.1037/a0025629. [DOI] [PubMed] [Google Scholar]
  40. Garcia O’Hearn H, Margolin G, John RS. Mothers’ and fathers’ reports of children’s reactions to naturalistic marital conflict. Journal of the American Academy of Child & Adolescent Psychiatry. 1997;36:1366–1373. doi: 10.1097/00004583-199710000-00018. [DOI] [PubMed] [Google Scholar]
  41. Gilbert P. Evolutionary approaches to psychopathology: the role of natural defences. Australian & New Zealand Journal of Psychiatry. 2001;35:17–27. doi: 10.1046/j.1440-1614.2001.00856.x. [DOI] [PubMed] [Google Scholar]
  42. Goodman R. The extended version of the Strengths and Difficulties Questionnaire as a guide to child psychiatric caseness and consequent burden. Journal of Child Psychology and Psychiatry. 1999;40:791–799. doi: 10.1111/1469-7610.00494. [DOI] [PubMed] [Google Scholar]
  43. Gordis EB, Margolin G, John RS. Parents’ hostility in dyadic marital and triadic family settings and children’s behavior problems. Journal of Consulting & Clinical Psychology. 2001;69:727–734. doi: 10.1037/0022006X.69.4.727. [DOI] [PubMed] [Google Scholar]
  44. Grych JH. Children’s appraisals of interparental conflict: Situational and contextual influences. Journal of Family Psychology. 1998;12:437–453. doi: 10.1037/0893-3200.12.3.437. [DOI] [Google Scholar]
  45. Grych JH. Marital relationships and parenting. In: Bornstein MH, editor. Handbook of parenting: Vol. 4: Social conditions and applied parenting. 2. Mahwah, NJ: Lawrence Erlbaum Associates Publishers; 2002. pp. 203–225. [Google Scholar]
  46. Grych JH, Fincham FD. Marital conflict and children’s adjustment: A cognitive-contextual framework. Psychological Bulletin. 1990;111:434–454. doi: 10.1037/0033-2909.108.2.267. [DOI] [PubMed] [Google Scholar]
  47. Johnston J, Roseby V, Kuehnle K. In the name of the child: A developmental approach to understanding and helping children of conflict and violent divorce. New York, NY: Springer Publishing; 2009. [Google Scholar]
  48. Jouriles EN, Spiller L, Stephens N, McDonald R, Swank P. Variability in adjustment of children of battered women: The role of child appraisals of interparent conflict. Cognitive Therapy and Research. 2000;24:233–249. doi: 10.1023/A:1005402310180. [DOI] [Google Scholar]
  49. Jouriles EN, Rosenfield D, McDonald R, Mueller V. Child involvement in interparental conflict and child adjustment problems: A longitudinal study of violent families. Journal of Abnormal Child Psychology. 2014;42:693–704. doi: 10.1007/s1082-013-9821-1. [DOI] [PMC free article] [PubMed] [Google Scholar]
  50. Jurkovic GJ, Morrell R, Thirkield A. Assessment of childhood parentification: Guidelines for researchers and clinicians. In: Chase N, editor. Burdened children: Theory, research, and treatment of parentification. Thousand Oaks, CA: Sage; 1999. pp. 92–113. [Google Scholar]
  51. Kerig PK. Assessing the links between interparental conflict and child adjustment: The conflicts and problem-solving scales. Journal of Family Psychology. 1996;10:454–473. doi: 10.1037/0893-3200.10.4.454. [DOI] [Google Scholar]
  52. King DW, King LA, McArdle JJ, Shalev AY, Doron-LaMarca S. Sequential temporal dependencies in associations between symptoms of depression and posttraumatic stress disorder: An application of bivariate latent difference score structural equation modeling. Multivariate Behavioral Research. 2009;44:437–464. doi: 10.1080/00273170903103308. [DOI] [PubMed] [Google Scholar]
  53. Kitzmann KM, Gaylord NK, Holt AR, Kenny ED. Child witnesses to domestic violence: A meta-analytic review. Journal of Consulting and Clinical Psychology. 2003;71:339–352. doi: 10.1037/0022-006X.71.2.339. [DOI] [PubMed] [Google Scholar]
  54. Kochanska G, Sanghag K, Barry RA, Philibert RA. Children’s genotypes interact with maternal responsive care in predicting competence: Diathesis-stress or differential susceptibility? Development and Psychopathology. 2011;23:605–616. doi: 10.1017/S0954579411000071. [DOI] [PMC free article] [PubMed] [Google Scholar]
  55. Luby JL, Heffelfinger AK, Mrakotsky C, Hessler MJ, Brown K, Hildebrand T. Preschool major depressive disorder (MDD): preliminary validation for developmentally modified DSM-IV criteria. Journal of the American Academy of Child & Adolescent Psychiatry. 2002;41:928–937. doi: 10.1097/00004583-200208000-00011. [DOI] [PubMed] [Google Scholar]
  56. Luby JL, Mrakotsky C, Heffelfinger A, Brown K, Spitznagel E. Characteristics of depressed preschoolers with and without anhedonia: Evidence for a melancholic depressive subtype in young children. The American Journal of Psychiatry. 2004;161:1998–2004. doi: 10.1176/appi.ajp.161.11.1998. [DOI] [PubMed] [Google Scholar]
  57. Luthar SS, Cicchetti D, Becker B. The construct of resilience: A critical evaluation and guidelines for future work. Child Development. 2000;71:543–562. doi: 10.1111/1467-8624.00164. [DOI] [PMC free article] [PubMed] [Google Scholar]
  58. McArdle JJ. Latent variable modeling of differences and changes with longitudinal data. Annual Review of Psychology. 2009;60:577–605. doi: 10.1146/annurev.psych.60.110707.163612. [DOI] [PubMed] [Google Scholar]
  59. McConnell MC, Kerig PK. Assessing coparenting in families of school-agee children: Validation of the coparenting and family rating system. Canadian Journal of Behavioural Science. 2002;34:44–58. [Google Scholar]
  60. Minuchin S. Families and family therapy. Cambridge, MA: Harvard University Press; 1974. [Google Scholar]
  61. Porter B, O’Leary K. Marital discord and childhood behavior problems. Journal of Abnormal Child Psychology. 1980;8:287–295. doi: 10.1007/BF00916376. [DOI] [PubMed] [Google Scholar]
  62. Rhoades KA. Children’s responses to interparental conflict: A meta-analysis of their associations with child adjustment. Child Development. 2008;79:1942–1956. doi: 10.1111/j.1467-8624.2008.01235.x. [DOI] [PMC free article] [PubMed] [Google Scholar]
  63. Roisman GI, Newman DA, Fraley R, Haltigan JD, Groh AM, Haydon KC. Distinguishing differential susceptibility from diathesis-stress: Recommendations for evaluating interaction effects. Development and Psychopathology. 2012;24:389–409. doi: 10.1017/S0954579412000065. [DOI] [PubMed] [Google Scholar]
  64. Sandler IN, Tein JY, West SG. Coping, stress, and psychological symptoms of children of divorce: A cross-sectional and longitudinal study. Child Development. 1994;65:1744–1763. doi: 10.2307/1131291. [DOI] [PubMed] [Google Scholar]
  65. Schermerhorn AC, Chow SM, Cummings EM. Developmental family processes and interparental conflict: Patterns of micro-level influences. Developmental Psychology. 2010;46:869–885. doi: 10.1037/a0019662. [DOI] [PMC free article] [PubMed] [Google Scholar]
  66. Schermerhorn AC, Cummings AC, DeCarlo CA, Davies PT. Children’s influence in the marital relationship. Journal of Family Psychology. 2007;21:259–269. doi: 10.1037/0893-3200.21.2.259. [DOI] [PubMed] [Google Scholar]
  67. Shaw DS, Bell RQ, Gilliom M. A truly early starter model of antisocial behavior revisited. Clinical Child and Family Psychology Review. 2000;3:155–172. doi: 10.1023/A:1009599208790. [DOI] [PubMed] [Google Scholar]
  68. Shelton KH, Harold GT. Interparental conflict, negative parenting, and children’s adjustment: Bridging links between parents’ depression and children’s psychological distress. Journal of Family Psychology. 2008;22:712–724. doi: 10.1037/a0013515. [DOI] [PubMed] [Google Scholar]
  69. Widaman KF, Ferrer E, Conger RD. Factorial invariance within longitudinal structural equation models: Measuring the same construct across time. Child Development Perspectives. 2010;4:10–18. doi: 10.1111/j.1750-8606.2009.00110.x. [DOI] [PMC free article] [PubMed] [Google Scholar]

RESOURCES