Corrigendum to “Indirect regulation of PCSK9 gene in inflammatory response by Porphyromonas gingivalis infection” [Heliyon 5 (1) (January 2019) e01111]

In the original published version of this article, an error was present in figure 3. During the typesetting stage, an incorrect version of figure 3 was uploaded which was missing several figure panels which had been present at acceptance. The authors apologize for this mistake. A corrected version of Figure 3 is displayed below. Both the HTML and PDF versions of the article have been updated to correct the error.

Fig. 3. Changes in serum lipid levels following the administration of P. gingivalis and nucleic acid antigens: (A) total cholesterol (TC), (B) low-density lipoprotein cholesterol (LDL-C), (C) high-density lipoprotein cholesterol (HDL-C), and (D) triglycerides (TG). N = 5 mice per group. Results are expressed as means ± SEM. *, P < 0.05; **, P < 0.01. Analysis of serum cholesterol levels.: Correlations between the serum lipids and serum PCSK9 level or hepatic TNF-α gene expression in wild-type mice. (E) LDL-C versus PCSK9, (F) HDL-C levels versus PCSK9, (G) TC versus PCSK9, (H) LDL-C versus Tnfα, (I) HDL-C versus Tnfα, and (J) TC versus Tnfα. The ratio of each LDL-C fraction level to the total: (K) large (φ28.6 nm), (L) medium (φ25.5 nm), (M) small (φ23.0 nm), or (N) very small (φ16.7–20.7 nm). N = 5 mice per group. Results are expressed as means ± SEM. *, P < 0.05; **, P < 0.01.