Abstract
We expand on existing understandings of health disparities among middle-class African Americans by examining how the postsecondary educational context gives rise to the unequal distribution of health. We used panel data (1994–2009) from the National Longitudinal Study of Adolescent to Adult Health (Add Health) to estimate whether the risk of developing metabolic syndrome by midlife significantly differs for African Americans who attended Historically Black College or Universities (HBCUs) versus predominantly White institutions. We found that HBCU enrollment was associated with a 35% reduction in the odds of metabolic syndrome. Furthermore, we demonstrate that HBCU attendees who grew up in more segregated environments experienced the greatest reductions in the likelihood of developing metabolic syndrome. Our results underscore the important role that HBCUs play in the lives of African Americans and suggest their impacts go far beyond traditional benchmarks of socioeconomic achievement to include key health outcomes.
Keywords: African Americans, chronic disease, education, HBCUs, metabolic syndrome, racial disparities, racial segregation, social determinants of health
Abbreviations
- Add Health
National Longitudinal Study of Adolescent to Adult Health
- AHAA
Adolescent Health and Academic Achievement
- HBCU
Historically Black College or University
- IPEDS
Integrated Postsecondary Education Data System
- PWI
predominately White institution
Racial disparities in health exact a heavy toll on US society, in terms of human life as well as social and economic costs. African Americans can expect to live 5 fewer years at birth compared with Whites (1). Moreover, the years they are alive are often spent living with one or more chronic illnesses or serious disabilities (1, 2). A robust body of research that seeks to identify the underlying causes of these stark racial disparities in health exists (3); however, much remains unknown about the social conditions that give rise to health disparities among Black subpopulations. The present study seeks to expand our understanding of how health is unequally distributed among African Americans who attend college in drastically different settings. Specifically, we asked to what extent does enrollment at a Historically Black College or University (HBCU) as opposed to a predominantly White institution (PWI) influence the health of college-educated African Americans.
Despite having relatively high levels of socioeconomic status, the health of middle-class African Americans lags behind that of similarly situated Whites. In fact, Black/White health disparities are more pronounced among nonpoor subgroups (3). One mechanism through which the health of African Americans is likely to erode over time or “weather” is through exposure to discrimination (4). Recent research suggests that upwardly mobile African Americans are just as likely to experience instances of discrimination as they ascend the socioeconomic hierarchy (5). These factors, taken together, indicate that there are specific aspects of the social environment in which middle-class African Americans live, work, play, and pray that are detrimental to their health. Education is a social exposure that deserves further study to more fully understand why racial disparities in health among middle-class African Americans remain pronounced despite having access to key social and economic resources.
Education has long been an important predictor of health, so much so that it is considered a fundamental cause of health disparities (6). Educational attainment is consistently associated with better health across a wide range of outcomes (7). Yet most of what we know about the education/health gradient is limited by a singular focus on completed education as the exposure of interest. Other key aspects of the context in which schooling occurs should be rigorously examined. We argue that, in order to more fully understand how education differentially shapes the distribution of morbidity and mortality across race, researchers should examine the quality of the educational setting rather than focusing only on the quantity of educational attainment (8).
For African Americans, HBCUs provide a unique environment within which learning occurs in predominantly Black spaces. These institutions allow African-American students to pursue educational and occupational goals, develop important life skills, and form lasting friendships while being sheltered, at least somewhat, from racial discrimination. HBCU enrollees are less likely to be the only (or one of a few) African Americans on campus, thus reducing the harmful health effects of tokenism (9, 10). Finally, by attending HBCUs, Black students are more likely than their counterparts at PWIs to interact with faculty of color who can serve as mentors, which is particularly advantageous for first-generation college students (11).
HBCU enrollment has been shown to predict socioeconomic achievement (12, 13); however, the literature regarding the health effects of HBCU attendance is sparse, and much remains unknown due to a primary focus on health behaviors during college as endpoints of interest and a lack of adequate comparison groups of PWI enrollees (14, 15). The few existing studies that focus on long-term health outcomes provide preliminary evidence that HBCU enrollment is protective for health behaviors or mental health but do not offer much information regarding physical health (16–18). The present study was designed to fill this gap.
Not only is it important to consider the context in which higher education occurs to accurately assess its role in giving rise to health disparities, it is critical to recognize the importance of the timing during which this social exposure typically takes place. Emerging adulthood is the life-course period during which the successful “launching” of young adults typically happens. It is when most individuals experience independence from their parents for the first time (19) and when health behaviors are commonly solidified into lifelong habits (20). Peer effects become particularly influential during emerging adulthood as individuals move out of their childhood homes and into shared living arrangements (21). Thus, the choice to attend an HBCU rather than a PWI might prove to be a particularly strong predictor of health across the life course for African Americans because of when during the life course it occurs.
We also consider whether the association between HBCU enrollment and health is more pronounced for individuals who grow up in predominantly Black or White environments. Although racial residential segregation (hereafter: segregation) has been consistently linked to suboptimal health outcomes for African Americans (22, 23), it is not clear the extent to which this association is the result of socioeconomic divestment or racial clustering. This is a critical question worth investigating further given that individuals residing in more segregated neighborhoods experience less interpersonal discrimination (24), and the association between discrimination and health is contingent on the level of neighborhood segregation (25).
For young African Americans who spent their formative years in mostly White spaces, attending an HBCU might benefit their health by reducing the frequency of racial discrimination and/or tokenism, both of which have been shown to have a negative impact on health outcomes (9, 10, 26, 27). Alternatively, because predominantly Black schools and neighborhoods are often underresourced, are often overcrowded, and have less-experienced teachers (28, 29), individuals growing up in these environments might benefit, at least with respect to their health, from the intraracial connections and same-race mentoring that can be found at HBCUs.
To this end, the present study uses data from the National Longitudinal Study of Adolescent to Adult Health (Add Health) to answer 3 specific research questions. First, can African Americans who attend an HBCU as opposed to a PWI expect to experience better health in adulthood? Second, is the association between HBCU enrollment and adult health largely attributable to early-life disadvantage? Third, do the protective health effects of HBCU enrollment differ for African Americans who grew up in more- or less-segregated environments?
METHODS
Add Health is a nationally representative, prospective cohort study of US middle- and high-school students who were in grades 7–12 in 1994–1995 (wave I). Follow-up interviews were conducted in 1996 (wave II), 2001 (wave III), and 2008 (wave IV). Additional data concerning respondents’ schools, neighborhoods, and social networks were collected from school administrators, parents, and friends across multiple waves. Of the total sample surveyed in wave I (n = 20,745), 23% of respondents (n = 4,807) self-identify as Black or African American. Of all eligible Add Health respondents, 79% participated in the initial survey (wave I); 88.6%, 77.4%, and 80.3% of these respondents were reinterviewed in waves II, III, and IV, respectively. Corresponding response rates for Black participants were 88.3%, 74.8%, and 80.5%.
For the present study, we used data from waves I, III, and IV to estimate the association between HBCU attendance and subsequent health in adulthood among African Americans. HBCU attendance was assessed at wave III (2001), when respondents were 18–26 years old, while health was captured at wave IV (2008), when respondents were 24–34 years old. Sociodemographic characteristics concerning one’s family of origin and early-life exposures were measured at wave I (1995) when respondents were between the ages of 12 and 19 years.
Add Health respondents interviewed at wave I and enrolled in an institution of higher learning at wave III were eligible to have information from the Integrated Postsecondary Education Data System (IPEDS) incorporated as part of the Adolescent Health and Academic Achievement (AHAA) supplement (30). Of the 5,487 Add Health respondents who reported attending college at wave III, 4,703 provided information to obtain high-school transcripts, 4,171 supplied a valid institutional identifier for college, and 4,157 had IPEDS data successfully linked. Corresponding numbers for Black respondents were 1,214, 967, 848, and 842, respectively. Of the 842 Black Add Health participants with linked IPEDS data, 727 had valid information on dependent variables of interest. It is this subset of respondents who comprise the analytical sample of the present study. Sensitivity analyses revealed that our final sample (n = 727) did not significantly differ from the 842 Black college enrollees with linked IPEDS data or the 1,214 Black respondents who reported attending college at wave III.
One of the AHAA questions indicated whether the respondent’s postsecondary institution was an HBCU. Thus, this variable captured the current as opposed to the degree-granting university. The National Center for Education Statistics (NCES), which produces IPEDS data, defines HBCUs as “… institutions that were established prior to 1964 with the principal mission of educating Black Americans.” (31) Based on this information, we constructed a dichotomous measure to reflect enrollment in an HBCU versus a PWI. Respondents in our sample attended a total of 319 institutions of higher learning, with 273 being PWIs and 46 being HBCUs.
To capture health at wave IV, we constructed an indicator of metabolic syndrome similar to Gaydosh et al. (32). We assessed whether respondents were above a high-risk threshold on 5 common biomarkers of cardiovascular health: blood pressure, waist circumference, triglycerides, high-density lipoprotein, and glycated hemoglobin (HbA1c) levels. Additional information regarding these thresholds can be found in Web Table 1 (available at https://doi.org/10.1093/aje/kwaa245). We defined metabolic syndrome as having 3 or more high-risk biomarkers. Participants with missing values on a biomarker were coded as low-risk for that variable; therefore, our results represent conservative estimates of the association between HBCU enrollment and metabolic syndrome. Sensitivity analyses conducted with respondents who had complete data on all metabolic syndrome indicators (n = 599) produced similar findings.
We controlled for a number of potential confounders, all of which were captured at wave I, when respondents were in grades 7–12, unless otherwise specified. These included age (continuous), sex (male vs. female), census region (Northeast, Midwest, South, West), and urbanicity (urban, suburban, rural). To account for important institutional differences between HBCUs and PWIs, we included an indicator of public versus private college. We were not able to further disaggregate for-profit institutions due to small cell sizes. Although the AHAA supplement does not have reliable information concerning college-specific graduation rates, we adjusted for the differential likelihood of college completion across HBCU status using a dichotomous indicator coded as 1 if the respondent graduated from college by wave IV and 0 if he/she did not. We captured the respondent’s baseline health status using the following 3 variables: obesity (yes/no), diabetes diagnosis (yes/no), and fair/poor self-rated health (yes/no).
We incorporated additional control variables, all of which were multi-item measures, at wave I to capture early-life socioeconomic disadvantage at the family, school, and neighborhood levels. We assessed childhood family disadvantage using the following dichotomous indicators asked of the reporting parent: neither parent attended college, neither parent was employed in a skilled blue-collar or white-collar occupation, family welfare receipt, and parents not married. We assigned respondents a value of 1 for each affirmative answer and a value of 0 for each negative answer. School disadvantage indicators included grade retention rate, drop-out rate, average class size, and proportion of teachers without graduate degrees, as well as the proportion of students for whom the families received public assistance, parent(s) were unemployed, parent(s) did not finish high school, and parent(s) were unmarried. Neighborhood disadvantage measures were based on the census tract of residence and captured the proportion families below poverty, adults without high-school degree, female-headed households, vacant households, and the unemployment rate. We assigned respondents a value of 1 (high disadvantage) or 0 (low disadvantage) if their school or neighborhood fell within the top quartile of values among the full Add Health wave I sample.
We estimated a series of logistic regression models to determine the extent to which HBCU attendance was associated with metabolic syndrome in adulthood. We regressed the probability of metabolic syndrome on HBCU enrollment and a number of potential confounders:
where x represents a vector of additional covariates including age, sex, census region, urbanicity, private versus public college, college graduation, and baseline health status, as well as family, school, and neighborhood disadvantage. To determine whether the association between HBCU attendance and metabolic syndrome significantly differed for respondents who grew up in more segregated environments, we generated 2 interaction terms (HBCU*school % Black and HBCU*neighborhood % Black), each of which was entered into the following regression model separately:
where x represents a vector of the same set of additional covariates. Robust standard errors were calculated using the Huber/White correction and clustered at the school level.
Regression models were estimated without sampling weights given that 1) our research questions are specific to a distinct subgroup of respondents—college-educated Blacks—and should not be generalized to a nationally representative population of US adolescents; and 2) applying sampling weights to small, demographically distinct subsamples can introduce bias into regression estimates (33). Missing values for covariates were calculated using multiple imputation methods with chained equations. Following Von Hippel (34), we did not impute values for dependent variables of interest; doing so adds little in efficiency and can create biased estimates. None of the control variables exceeded 5% missing, and most fell below 1% missing.
RESULTS
Descriptive findings are presented in Table 1. Almost 30% of respondents had metabolic syndrome at wave IV. This result is remarkable because Add Health participants are still relatively young (24–34 years old), and it speaks to the health burden that college-educated African Americans face despite their relatively high socioeconomic status. The distribution of metabolic syndrome across institutions of higher learning is not equivalent, with 31% of PWI attendees but only 23% of HBCU attendees having this condition.
Table 1.
Full Sample (n = 727) | PWI (n = 551) | HBCU (n = 176) | |||||
---|---|---|---|---|---|---|---|
Variable | Proportion | Mean (SD) | Proportion | Mean (SD) | Proportion | Mean (SD) | T Statistic for Difference |
Metabolic syndrome | 0.29 | 0.31 | 0.23 | −1.93b | |||
HBCU vs. PWI | 0.32 | ||||||
School, proportion Black | 0.39 (0.27) | 0.34 (0.24) | 0.57 (0.28) | 10.81c | |||
Neighborhood, proportion Black | 0.50 (0.34) | 0.47 (0.34) | 0.59 (0.32) | 4.31c | |||
Age, years | 14.95 (1.69) | 15.08 (1.67) | 14.53 (1.66) | −3.75c | |||
Female sex | 0.64 | 0.66 | 0.58 | −1.95b | |||
Census region | |||||||
Northeast | 0.14 | 0.16 | 0.05 | −3.59c | |||
Midwest | 0.22 | 0.21 | 0.28 | 2.20d | |||
South | 0.19 | 0.23 | 0.03 | −5.89c | |||
West | 0.45 | 0.40 | 0.64 | 5.17c | |||
Urbanicity | |||||||
Urban | 0.53 | 0.52 | 0.57 | 1.25 | |||
Suburban | 0.28 | 0.30 | 0.22 | −2.14d | |||
Rural | 0.19 | 0.18 | 0.21 | 0.85 | |||
Private (vs. public) college | 0.21 | 0.18 | 0.30 | 3.52c | |||
Graduated college | 0.57 | 0.54 | 0.68 | 3.38c | |||
Childhood disadvantage | |||||||
Family | 1.27 (0.95) | 1.31 (0.98) | 1.13 (0.86) | −2.20d | |||
School | 2.77 (1.93) | 2.75 (1.92) | 2.85 (1.96) | 0.58 | |||
Neighborhood | 2.08 (1.90) | 1.92 (1.87) | 2.57 (1.91) | 4.12c | |||
Childhood health | |||||||
Obesity | 0.18 | 0.18 | 0.16 | −0.31 | |||
Diabetes | 0.09 | 0.10 | 0.09 | 0.00 | |||
Self-rated health | |||||||
Excellent | 0.33 | 0.34 | 0.32 | −0.34 | |||
Very good | 0.40 | 0.40 | 0.39 | −0.17 | |||
Good | 0.20 | 0.19 | 0.23 | 1.39 | |||
Fair or poor | 0.07 | 0.08 | 0.05 | −1.21 |
Abbreviations: HBCU, Historically Black College or University; PWI, predominately White institution; SD, standard deviation.
a Descriptive statistics generated using imputed values for covariates. P values are from 2-tailed tests.
b P < 0.1.
c P < 0.001.
d P < 0.05.
We also note important differences across HBCU status in the types of educational and neighborhood environments Black Add Health respondents spent time in during their teenage years. PWI enrollees went to high schools that were, on average, 34% Black, while their counterparts at HBCUs attended high schools that were, on average, 57% Black. Similarly, respondents at PWIs grew up in neighborhoods that were significantly less segregated (47% Black) than respondents at HBCUs (59% Black). Despite a greater likelihood of HBCU attendees to grow up in predominantly Black neighborhoods, we failed to find significant differences across HBCU status on 2 of the 3 measures of family disadvantage. Additional descriptive statistics for control variables are presented in Table 1. Individual indicators used to construct our measures of family, school, and neighborhood disadvantage are presented in Web Table 2.
Results from logistic regression models are presented in Table 2. Model 1 reveals the bivariate association between HBCU attendance and metabolic syndrome. HBCU enrollees faced 32% lower odds of developing metabolic syndrome compared with PWI enrollees. After controlling for all potential confounders, the magnitude of the odds ratio remains remarkably consistent and even becomes slightly more as opposed to less pronounced. Attending an HBCU versus a PWI is associated with a 35% reduction in the odds of having metabolic syndrome net of the effect of key covariates (model 7).
Table 2.
Model 1
(n = 727) |
Model 2
(n = 727) |
Model 3
(n = 727) |
Model 4
(n = 727) |
Model 5
(n = 727) |
Model 6
(n = 727) |
Model 7
(n = 727) |
||||||||
---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|
Independent Variables | OR | 95% CI | OR | 95% CI | OR | 95% CI | OR | 95% CI | OR | 95% CI | OR | 95% CI | OR | 95% CI |
HBCU vs. PWI | 0.68b | 0.47,0.99 | 0.61c | 0.42,0.88 | 0.63b | 0.44,0.91 | 0.64b | 0.44,0.93 | 0.63b | 0.44,0.89 | 0.63b | 0.45,0.90 | 0.65b | 0.45,0.94 |
Age | 1.01 | 0.91,1.09 | 1.00 | 0.91,1.09 | 1.00 | 0.91,1.09 | 0.98 | 0.90,1.08 | 0.97 | 0.89,1.07 | 0.98 | 0.89,1.07 | ||
Female sex | 0.91 | 0.67,1.24 | 0.93 | 0.68,1.26 | 0.92 | 0.68,1.25 | 0.91 | 0.67,1.23 | 0.93 | 0.68,1.26 | 0.94 | 0.66,1.35 | ||
Census region | ||||||||||||||
Northeast | 1.00 | Referent | 1.00 | Referent | 1.00 | Referent | 1.00 | Referent | 1.00 | Referent | 1.00 | Referent | ||
Midwest | 0.53b | 0.30,0.94 | 0.54b | 0.31,0.93 | 0.54b | 0.31,0.92 | 0.57b | 0.34,0.94 | 0.56b | 0.34,0.93 | 0.64 | 0.38,1.09 | ||
South | 0.81 | 0.50,1.29 | 0.81 | 0.51,1.28 | 0.81 | 0.51,1.29 | 0.82 | 0.52,1.29 | 0.82 | 0.52,1.30 | 0.86 | 0.54,1.38 | ||
West | 0.75 | 0.52,1.08 | 0.76 | 0.52,1.10 | 0.77 | 0.53,1.11 | 0.75d | 0.55,1.03 | 0.68b | 0.47,0.97 | 0.71d | 0.50,1.00 | ||
Urbanicity | ||||||||||||||
Urban | 1.00 | Referent | 1.00 | Referent | 1.00 | Referent | 1.00 | Referent | 1.00 | Referent | 1.00 | Referent | ||
Suburban | 0.96 | 0.62,1.49 | 0.95 | 0.61,1.48 | 0.96 | 0.62,1.48 | 0.98 | 0.64,1.48 | 0.93 | 0.61,1.42 | 1.01 | 0.65,1.55 | ||
Rural | 0.93 | 0.60,1.43 | 0.93 | 0.61,1.43 | 0.92 | 0.60,1.42 | 0.92 | 0.60,1.43 | 0.93 | 0.60,1.43 | 0.91 | 0.59,1.41 | ||
Private (vs. public) college | 1.19 | 0.83,1.71 | 1.18 | 0.82,1.70 | 1.16 | 0.81,1.68 | 1.14 | 0.79,1.65 | 1.20 | 0.82,1.75 | ||||
Graduated college | 0.65b | 0.47,0.92 | 0.67b | 0.47,0.95 | 0.68b | 0.48,0.97 | 0.68b | 0.48,0.96 | 0.70b | 0.50,0.98 | ||||
Family disadvantage | 1.07 | 0.87,1.30 | 1.05 | 0.86,1.28 | 1.06 | 0.86,1.29 | 1.06 | 0.87,1.29 | ||||||
School disadvantage | 1.06 | 0.99,1.15 | 1.08d | 1.00,1.17 | 1.07 | 0.98,1.16 | ||||||||
Neighborhood disadvantage | 0.94 | 0.85,1.03 | 0.94 | 0.85,1.03 | ||||||||||
Childhood health | ||||||||||||||
Obesity | 2.47e | 1.47,4.16 | ||||||||||||
Diabetes | 1.30 | 0.59,2.88 | ||||||||||||
Self-rated health | 0.98 | 0.82,1.17 |
Abbreviations: CI, confidence interval; HBCU, Historically Black College or University; OR, odds ratio; PWI, predominately White institution.
a Each model progressively incorporates additional control variables, sociodemographic measures, postsecondary institution and college attainment measures, early-life disadvantage measures, and adolescent health measures. P values are from 2-tailed tests.
b P < 0.05.
c P < 0.01.
d P < 0.1.
e P < 0.001.
We used results from logistic regression models with interaction terms (HBCU*% Black), found in Web Table 3, to calculate the average marginal effects of HBCU attendance on the predicted probability of metabolic syndrome across levels of school (Figure 1) or neighborhood (Figure 2) segregation. Percentile Black is shown on the x-axis, while the difference in the probability of metabolic syndrome between HBCU and PWI enrollees is displayed on the y-axis. For Black respondents who attended predominantly White high schools (5th percentile), those enrolled in either HBCUs or PWIs were similarly likely to develop metabolic syndrome (Figure 1). The health protective effect of HBCU attendance becomes more evident as the proportion of Black students in a given high school increases, although the magnitude of this change is modest and does not reach statistical significance. HBCU enrollees who attended integrated (50th percentile) or predominantly Black (95th percentile) high schools were 8.1 and 12.5 percentage points less likely to develop metabolic syndrome than PWI enrollees, respectively.
A similar, and more pronounced, trend whereby the association between HBCU enrollment and health is more evident at higher levels of segregation, can be seen in Figure 2. Black Add Health respondents who grew up in predominantly White (5th percentile) neighborhoods were similarly likely to develop metabolic syndrome regardless of the type of college attended. The opposite is true for HBCU enrollees who lived in integrated (50th percentile) or predominantly Black (95th percentile) neighborhoods. For these respondents, matriculating at an HBCU versus a PWI was associated with a 7.5 and 16.4 percentage-point reduction in the risk of metabolic syndrome, respectively.
DISCUSSION
Our empirical findings suggest that in addition to providing African Americans with a unique setting in which to pursue a college degree and serving as a driver of upward mobility, HBCUs offer Black students important, long-term health benefits. We report that African Americans who attend HBCUs were significantly less likely than those at PWIs to develop metabolic syndrome in adulthood. The magnitude of this health benefit is similar to those reported in the literature after engaging in comprehensive changes to diet and exercise regimens (35). This finding is also notable because metabolic syndrome is a precursor to heart disease and stroke—2 chronic diseases that account for a substantial proportion of mortality in the United States and that serve as key drivers of racial disparities in health (36). Given the underlying pathophysiological mechanisms that link exposures to race-related stressors to suboptimal health outcomes primarily via the relentless triggering of the human stress response (37), we expect the potential health benefits of HBCU attendance not to be limited to this one outcome.
Unfortunately, we cannot discern why enrolling in an HBCU was associated with a lower risk of metabolic syndrome for African Americans, but we can hypothesize. The health benefits of HBCU attendance might stem from an educational environment that 1) more effectively shields students from exposure to racial discrimination; 2) allows African-American college students to focus on learning and academic mastery while avoiding race-related stressors that have been linked to poor health outcomes, such as tokenism (10) and hypervigilance (38); or 3) provides ample access to same-race mentors who have successfully navigated predominantly White environments in pursuit of educational or occupational advancement. Exploring how discrimination might mediate the association between HBCU attendance and health is important given that African Americans, unlike members of other racial/ethnic groups, continue to face discrimination even as they experience upward socioeconomic mobility (5), and the health impacts of discrimination are likely to reverberate over time, both within and across generations (39, 40).
One of the most consequential findings of the present study is that the association between HBCU attendance and health appears to operate differently for those who grew up in more- or less-segregated environments. We note that the difference in the probability of metabolic syndrome was most pronounced for HBCU enrollees who went to predominantly Black high schools or lived in predominantly Black neighborhoods. Due to data constraints, we cannot identify the underlying mechanisms that produced these patterns, but these results suggest that HBCUs are likely to be health protective for the segment of society who needs it most—those growing up in the most racially isolated environments. Moreover, this finding underscores the important role that place, in general, and segregation, specifically, plays in the unequal distribution of health, and that its influence gives rise to health disparities not only between races but also among members of a single race (22, 41).
Our results should be considered in light of several limitations. First, our analytical sample is small and limits the amount of statistical power available to detect differential health outcomes by HBCU status. Second, not all Add Health respondents who attended college at wave III had valid, linked IPEDS data as part of the AHAA supplement, thus it remains to be seen whether our results are generalizable to all African-American college students. However, sensitivity analyses did not reveal significant differences between these 2 subsamples. Third, although we were able to follow respondents into adulthood, we do not know whether or how their health fluctuates as they move through successive life-course stages. Fourth, although we control for potential confounders, doing so cannot fully account for unobserved heterogeneity between HBCU and PWI enrollees. Prior research has shown that African Americans from disadvantaged backgrounds with lower levels of school achievement are more likely to attend HBCUs (11); therefore, our results might be conservative estimates of the true association between HBCU enrollment and health. Finally, we wanted to further disaggregate analyses by sex but could not do so with much precision due to our limited sample size. We look to future research to examine whether the association between HBCU enrollment and health differs for men and women.
In order to gain a deeper understanding of how racial inequalities in health shape the life chances of racial minorities in the United States, researchers need to stop treating African Americans as a monolithic group and start examining important differences among them. The present study sought to do this by 1) focusing on differential health outcomes among college-educated Blacks; and 2) asking whether the specific context in which higher education occurs has lasting health benefits. We find that for African Americans, attending an HBCU as opposed to a PWI reduces the risk of developing metabolic syndrome by adulthood, particularly for individuals who grew up in segregated environments. These results lend credence to the idea that it is not just the information that is conveyed within a classroom setting that makes education a valuable social resource able to influence multiple outcomes, including health. To this end, policy makers should consider the full range of benefits that HBCUs are likely to have on the health and well-being of their students, both upon graduation and as they move through subsequent phases of their lives, as well as recognizing them as a force for change in combatting racial inequalities in health.
Supplementary Material
ACKNOWLEDGMENTS
Author affiliations: Department of Sociology, Ohio State University, Columbus, Ohio, United States (Cynthia G. Colen, Nicolo P. Pinchak); Division of Health Behavior and Health Promotion, College of Public Health, Ohio State University, Columbus, Ohio, United States (Cynthia G. Colen, Kierra S. Barnett); Institute for Population Research, Ohio State University, Columbus, Ohio, United States (Cynthia G. Colen, Nicolo P. Pinchak, Kierra S. Barnett); and Kirwan Institute for the Study of Race and Ethnicity, Ohio State University, Columbus, Ohio, United States (Kierra S. Barnett).
This research was supported, in part, by the Eunice Kennedy Shriver National Institute of Child Health and Human Development (grant P2C-HD058484) awarded to the Ohio State University Institute for Population Research. N.P.P.'s effort was supported by the National Science Foundation Graduate Research Fellowship Program (grant DGE1255832). Data for this study are from Add Health, a program funded by the Eunice Kennedy Shriver National Institute of Child Health and Human Development (grant P01-HD31921), with cooperative support from 23 other federal agencies and foundations. The Adolescent Health and Academic Achievement (AHAA) study was funded by grants from the Eunice Kennedy Shriver National Institute of Child Health and Human Development (grant R01 HD040428-02, Chandra Muller, PI; 5 R24 HD042849, Population Research Center, University of Texas at Austin) and National Science Foundation (grant REC-0126167, Chandra Muller, PI, and Pedro Reyes, Co-PI).
The opinions and conclusions expressed herein are solely those of the authors and should not be construed as representing the opinions or policy of any agency of the federal government.
Conflict of interest: none declared.
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